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TrkB-ICD fragment, originating from BDNF receptor cleavage, is translocated to ...

Fonseca-Gomes, João; Jerónimo-Santos, André; Lesnikova, Angelina; Casarotto, Plinio; Castrén, Eero; Sebastião, Ana M; Diógenes, Maria José

The signaling of brain-derived neurotrophic factor (BDNF) has been suggested to be impaired in Alzheimer's disease (AD), which may compromise the function of BDNF upon neuronal activity and survival. Accordingly, decreased levels of BDNF and its tropomyosin-receptor kinase B-full-length (TrkB-FL) have been detected in human brain samples of AD patients. We have previously found that neuronal exposure to amyloid...

Date: 2019   |   Origin: Repositório da Universidade de Lisboa
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