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Uremic bleeding : pathogenesis and therapy

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Summary:Background: We reviewed current understanding of the pathophysiology of the uremic bleeding diathesis and discuss accepted therapeutic interventions that minimize the risk of bleeding in the uremic patient. Methods: Computerized literature searches and references from previous publications, including articles describing original research and reviews pertaining to the pathophysiology of and clinical approach to uremic bleeding. Results: The most common hemorrhagic manifestations in uremia are prolonged bleeding from puncture sites; nasal, gastrointestinal and genitourinary bleeding; and subdural hematomas. The most useful clinical laboratory test to assess both bleeding risk and response to therapy is bleeding time. It correlates better with clinical bleeding complications than indices of azotemia (eg, blood urea nitrogen [BUN], creatinine) or in vitro platelet aggregation tests. A low hematocrit is also correlated with increased bleeding risk. Anemia plays an important role in the bleeding diathesis of uremia and its correction with red cell transfusions or human recombinant erythropoietin is critical. Anticoagulation during hemodialysis may transiently exacerbate the bleeding diathesis. Hemodialysis and peritoneal dialysis improve the hemostatic defect and renal transplantation totally corrects it. Cryoprecipitate has been largely replaced by desmopressin acetate, which acts promptly (in less than 1 hour) but has a short duration of action (hours) and exhibits tachyphylaxis. Conjugated estrogens are slower in the onset of action (about 6 hours) but their effect lasts for about 2 weeks. Conclusions: The pathophysiology of the bleeding diathesis of uremia is complex and incompletely understood but useful clinical tests and therapies have evolved empirically. Broadly available dialysis and the advent of erythropoietin are likely to reduce the magnitude of this problem.
Main Authors:Weigert, André L.
Other Authors:Schafer, Andrew I.
Subject:Uremic bleeding diathesis Anemia Erythropoietin
Year:1998
Country:Portugal
Document type:article
Access type:restricted access
Associated institution:Universidade de Lisboa
Language:English
Origin:Repositório da Universidade de Lisboa
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author Weigert, André L.
author2 Schafer, Andrew I.
author2_role author
author_facet Weigert, André L.
Schafer, Andrew I.
author_role author
contributor_name_str_mv Repositório Científico de Acesso Aberto da ULisboa
country_str PT
creators_json_txt [{\"Person.name\":\"Weigert, André L.\"},{\"Person.name\":\"Schafer, Andrew I.\"}]
datacite.contributors.contributor.contributorName.fl_str_mv Repositório Científico de Acesso Aberto da ULisboa
datacite.creators.creator.creatorName.fl_str_mv Weigert, André L.
Schafer, Andrew I.
datacite.date.Accepted.fl_str_mv 1998-08-01T00:00:00Z
datacite.date.available.fl_str_mv 2021-01-22T11:00:44Z
datacite.date.embargoed.fl_str_mv 2021-01-22T11:00:44Z
datacite.rights.fl_str_mv http://purl.org/coar/access_right/c_16ec
datacite.subjects.subject.fl_str_mv Uremic bleeding diathesis
Anemia
Erythropoietin
datacite.titles.title.fl_str_mv Uremic bleeding : pathogenesis and therapy
dc.contributor.none.fl_str_mv Repositório Científico de Acesso Aberto da ULisboa
dc.creator.none.fl_str_mv Weigert, André L.
Schafer, Andrew I.
dc.date.Accepted.fl_str_mv 1998-08-01T00:00:00Z
dc.date.available.fl_str_mv 2021-01-22T11:00:44Z
dc.date.embargoed.fl_str_mv 2021-01-22T11:00:44Z
dc.format.none.fl_str_mv application/pdf
dc.identifier.none.fl_str_mv http://hdl.handle.net/10451/45901
dc.language.none.fl_str_mv eng
dc.publisher.none.fl_str_mv Elsevier
dc.rights.none.fl_str_mv http://purl.org/coar/access_right/c_16ec
dc.subject.none.fl_str_mv Uremic bleeding diathesis
Anemia
Erythropoietin
dc.title.fl_str_mv Uremic bleeding : pathogenesis and therapy
dc.type.none.fl_str_mv http://purl.org/coar/resource_type/c_6501
description Background: We reviewed current understanding of the pathophysiology of the uremic bleeding diathesis and discuss accepted therapeutic interventions that minimize the risk of bleeding in the uremic patient. Methods: Computerized literature searches and references from previous publications, including articles describing original research and reviews pertaining to the pathophysiology of and clinical approach to uremic bleeding. Results: The most common hemorrhagic manifestations in uremia are prolonged bleeding from puncture sites; nasal, gastrointestinal and genitourinary bleeding; and subdural hematomas. The most useful clinical laboratory test to assess both bleeding risk and response to therapy is bleeding time. It correlates better with clinical bleeding complications than indices of azotemia (eg, blood urea nitrogen [BUN], creatinine) or in vitro platelet aggregation tests. A low hematocrit is also correlated with increased bleeding risk. Anemia plays an important role in the bleeding diathesis of uremia and its correction with red cell transfusions or human recombinant erythropoietin is critical. Anticoagulation during hemodialysis may transiently exacerbate the bleeding diathesis. Hemodialysis and peritoneal dialysis improve the hemostatic defect and renal transplantation totally corrects it. Cryoprecipitate has been largely replaced by desmopressin acetate, which acts promptly (in less than 1 hour) but has a short duration of action (hours) and exhibits tachyphylaxis. Conjugated estrogens are slower in the onset of action (about 6 hours) but their effect lasts for about 2 weeks. Conclusions: The pathophysiology of the bleeding diathesis of uremia is complex and incompletely understood but useful clinical tests and therapies have evolved empirically. Broadly available dialysis and the advent of erythropoietin are likely to reduce the magnitude of this problem.
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person_str_mv Weigert, André L.
Schafer, Andrew I.
publishDate 1998
publisher.none.fl_str_mv Elsevier
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spelling engElsevierpt_PTBackground: We reviewed current understanding of the pathophysiology of the uremic bleeding diathesis and discuss accepted therapeutic interventions that minimize the risk of bleeding in the uremic patient. Methods: Computerized literature searches and references from previous publications, including articles describing original research and reviews pertaining to the pathophysiology of and clinical approach to uremic bleeding. Results: The most common hemorrhagic manifestations in uremia are prolonged bleeding from puncture sites; nasal, gastrointestinal and genitourinary bleeding; and subdural hematomas. The most useful clinical laboratory test to assess both bleeding risk and response to therapy is bleeding time. It correlates better with clinical bleeding complications than indices of azotemia (eg, blood urea nitrogen [BUN], creatinine) or in vitro platelet aggregation tests. A low hematocrit is also correlated with increased bleeding risk. Anemia plays an important role in the bleeding diathesis of uremia and its correction with red cell transfusions or human recombinant erythropoietin is critical. Anticoagulation during hemodialysis may transiently exacerbate the bleeding diathesis. Hemodialysis and peritoneal dialysis improve the hemostatic defect and renal transplantation totally corrects it. Cryoprecipitate has been largely replaced by desmopressin acetate, which acts promptly (in less than 1 hour) but has a short duration of action (hours) and exhibits tachyphylaxis. Conjugated estrogens are slower in the onset of action (about 6 hours) but their effect lasts for about 2 weeks. Conclusions: The pathophysiology of the bleeding diathesis of uremia is complex and incompletely understood but useful clinical tests and therapies have evolved empirically. Broadly available dialysis and the advent of erythropoietin are likely to reduce the magnitude of this problem.application/pdfpt_PTUremic bleeding : pathogenesis and therapyWeigert, André L.Schafer, Andrew I.HostingInstitutionOrganizationalRepositório Científico de Acesso Aberto da ULisboae-mailmailto:repositorio@reitoria.ulisboa.ptrepositorio@reitoria.ulisboa.ptISSNIsPartOf0002-9629DOIIsPartOf10.1097/00000441-199808000-000052021-01-22T11:00:44Z1998-081998-08-01T00:00:00ZHandlehttp://hdl.handle.net/10451/45901http://purl.org/coar/access_right/c_16ecrestricted accessUremic bleeding diathesisAnemiaErythropoietin1835713 bytesliteraturehttp://purl.org/coar/resource_type/c_6501journal articlehttp://purl.org/coar/access_right/c_16ecapplication/pdffulltexthttps://repositorio.ulisboa.pt/bitstreams/8cc28929-ac64-4ecc-b08d-c2232f2c2c45/downloadThe American Journal of the Medical Sciences316294104
spellingShingle Uremic bleeding : pathogenesis and therapy
Weigert, André L.
Uremic bleeding diathesis
Anemia
Erythropoietin
status SINGLETON
subject.fl_str_mv Uremic bleeding diathesis
Anemia
Erythropoietin
title Uremic bleeding : pathogenesis and therapy
title_full Uremic bleeding : pathogenesis and therapy
title_fullStr Uremic bleeding : pathogenesis and therapy
title_full_unstemmed Uremic bleeding : pathogenesis and therapy
title_short Uremic bleeding : pathogenesis and therapy
title_sort Uremic bleeding : pathogenesis and therapy
topic Uremic bleeding diathesis
Anemia
Erythropoietin
topic_facet Uremic bleeding diathesis
Anemia
Erythropoietin
url http://hdl.handle.net/10451/45901
visible 1