Autor(es): de Oliveira, Raquel ; Almeida, Manuel ; Lavado, Pedro ; Baptista, Alexandre
Data: 2024
Identificador Persistente: http://hdl.handle.net/10400.1/25937
Origem: Sapientia - Universidade do Algarve
Assunto(s): Chemical and Drug Induced Liver Injury; Diabetic Ketoa-cidosis; Pantoprazole; Cetoacidose Diabética; Lesões Hepáticas Induzidas por Produtos Químicos e Medicamentos; Pantoprazol
Critically ill patients are at higher risk of acquired liver in-jury, given the multiple coexisting potential causes of injury.1They are also at risk of stress ulcers, and prophylaxis with proton pump inhibitors (PPIs) is common in Intensive Care Units (ICUs). A 54-year-old woman was admitted to the ICU due to diabetic ketoacidosis (DKA). On admission, she was hemo-dynamically stable, with a Glasgow Coma Scale score of 7 (E2V1M4). Her abdominal examination was normal, without palpable organomegalies, and her liver blood tests were within the normal range. She was intubated for airway pro-tection and started on intravenous fluids, insulin perfusion, and prophylaxis with intravenous pantoprazole 40 mg/day.
