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2,3-Diarylxanthones as potential inhibitors of Arachidonic acid metabolic pathways

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Resumo:In response to an inflammatory stimulus, arachidonic acid (AA), the main polyunsaturated fatty acid present in the phospholipid layer of cell membranes, is released and metabolized to a series of eicosanoids. These bioactive lipid mediators of inflammation arise physiologically through the action of the enzymes 5-lipoxygenase (5-LOX) and cyclooxygenases (constitutive COX-1 and inducible COX-2). It is believed that dual inhibition of 5-LOX and COXs may have a higher beneficial impact in the treatment of inflammatory disorders rather than the inhibition of each enzyme. With this demand for new dual-acting anti-inflammatory agents, a range of 2,3-diarylxanthones were tested through their ability to interact in the AA metabolism. In vitro anti-inflammatory activity was evaluated through the inhibition of 5-LOX-catalyzed leukotriene B4 (LTB4) formation in human neutrophils and inhibition of COX-1- and COX-2-catalyzed prostaglandin E2 (PGE2) formation in human whole blood. The results showed that some of the studied arylxanthones were able to prevent LTB4 production in human neutrophils, in a concentration-dependent manner. The xanthone with a 2-catechol was the most active one (IC50 ∼ 9 μM). The more effective arylxanthones in preventing COX-1-catalyzed PGE2 production presented IC50 values from 1 to 7 μM, exhibiting a structural feature with at least one non-substituted aryl group. All the studied arylxanthones were ineffective to prevent the formation of PGE2 catalyzed by COX-2, up to the maximum concentration of 100 μM. The ability of the tested 2,3-diarylxanthones to interact with both 5-LOX and COX-1 pathways constitutes an important step in the research of novel dual-acting anti-inflammatory drugs.
Autores principais:Santos, Clementina M.M.
Outros Autores:Ribeiro, Daniela; Silva, Artur; Fernandes, Eduarda
Assunto:Xanthones 5-LOX COX-1 COX-2 Human neutrophils Human whole-blood assays
Ano:2017
País:Portugal
Tipo de documento:artigo
Tipo de acesso:acesso aberto
Instituição associada:Instituto Politécnico de Bragança
Idioma:inglês
Origem:Biblioteca Digital do IPB
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author Santos, Clementina M.M.
author2 Ribeiro, Daniela
Silva, Artur
Fernandes, Eduarda
author2_role author
author
author
author_facet Santos, Clementina M.M.
Ribeiro, Daniela
Silva, Artur
Fernandes, Eduarda
author_role author
contributor_name_str_mv Biblioteca Digital do IPB
country_str PT
creators_json_txt [{\"Person.name\":\"Santos, Clementina M.M.\",\"Person.identifier.orcid\":\"0000-0003-4380-7990\"},{\"Person.name\":\"Ribeiro, Daniela\"},{\"Person.name\":\"Silva, Artur\"},{\"Person.name\":\"Fernandes, Eduarda\"}]
datacite.contributors.contributor.contributorName.fl_str_mv Biblioteca Digital do IPB
datacite.creators.creator.creatorName.fl_str_mv Santos, Clementina M.M.
Ribeiro, Daniela
Silva, Artur
Fernandes, Eduarda
datacite.date.Accepted.fl_str_mv 2017-01-01T00:00:00Z
datacite.date.available.fl_str_mv 2017-09-22T10:59:41Z
datacite.date.embargoed.fl_str_mv 2017-09-22T10:59:41Z
datacite.rights.fl_str_mv http://purl.org/coar/access_right/c_abf2
datacite.subjects.subject.fl_str_mv Xanthones
5-LOX
COX-1
COX-2
Human neutrophils
Human whole-blood assays
datacite.titles.title.fl_str_mv 2,3-Diarylxanthones as potential inhibitors of Arachidonic acid metabolic pathways
dc.contributor.none.fl_str_mv Biblioteca Digital do IPB
dc.creator.none.fl_str_mv Santos, Clementina M.M.
Ribeiro, Daniela
Silva, Artur
Fernandes, Eduarda
dc.date.Accepted.fl_str_mv 2017-01-01T00:00:00Z
dc.date.available.fl_str_mv 2017-09-22T10:59:41Z
dc.date.embargoed.fl_str_mv 2017-09-22T10:59:41Z
dc.format.none.fl_str_mv application/pdf
dc.identifier.none.fl_str_mv http://hdl.handle.net/10198/14519
dc.language.none.fl_str_mv eng
dc.publisher.none.fl_str_mv Springer Verlag
dc.rights.none.fl_str_mv http://purl.org/coar/access_right/c_abf2
dc.subject.none.fl_str_mv Xanthones
5-LOX
COX-1
COX-2
Human neutrophils
Human whole-blood assays
dc.title.fl_str_mv 2,3-Diarylxanthones as potential inhibitors of Arachidonic acid metabolic pathways
dc.type.none.fl_str_mv http://purl.org/coar/resource_type/c_6501
description In response to an inflammatory stimulus, arachidonic acid (AA), the main polyunsaturated fatty acid present in the phospholipid layer of cell membranes, is released and metabolized to a series of eicosanoids. These bioactive lipid mediators of inflammation arise physiologically through the action of the enzymes 5-lipoxygenase (5-LOX) and cyclooxygenases (constitutive COX-1 and inducible COX-2). It is believed that dual inhibition of 5-LOX and COXs may have a higher beneficial impact in the treatment of inflammatory disorders rather than the inhibition of each enzyme. With this demand for new dual-acting anti-inflammatory agents, a range of 2,3-diarylxanthones were tested through their ability to interact in the AA metabolism. In vitro anti-inflammatory activity was evaluated through the inhibition of 5-LOX-catalyzed leukotriene B4 (LTB4) formation in human neutrophils and inhibition of COX-1- and COX-2-catalyzed prostaglandin E2 (PGE2) formation in human whole blood. The results showed that some of the studied arylxanthones were able to prevent LTB4 production in human neutrophils, in a concentration-dependent manner. The xanthone with a 2-catechol was the most active one (IC50 ∼ 9 μM). The more effective arylxanthones in preventing COX-1-catalyzed PGE2 production presented IC50 values from 1 to 7 μM, exhibiting a structural feature with at least one non-substituted aryl group. All the studied arylxanthones were ineffective to prevent the formation of PGE2 catalyzed by COX-2, up to the maximum concentration of 100 μM. The ability of the tested 2,3-diarylxanthones to interact with both 5-LOX and COX-1 pathways constitutes an important step in the research of novel dual-acting anti-inflammatory drugs.
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oai_identifier_str oai:bibliotecadigital.ipb.pt:10198/14519
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person_str_mv Santos, Clementina M.M.
Santos, Clementina M.M.
https://www.ciencia-id.pt/9018-DB9C-C590
9018-DB9C-C590
http://orcid.org/0000-0003-4380-7990
0000-0003-4380-7990
Ribeiro, Daniela
Silva, Artur
Fernandes, Eduarda
publishDate 2017
publisher.none.fl_str_mv Springer Verlag
reponame_str Biblioteca Digital do IPB
repository_id_str urn:repositoryAcronym:ipb
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spelling engSpringer Verlagpt_PTIn response to an inflammatory stimulus, arachidonic acid (AA), the main polyunsaturated fatty acid present in the phospholipid layer of cell membranes, is released and metabolized to a series of eicosanoids. These bioactive lipid mediators of inflammation arise physiologically through the action of the enzymes 5-lipoxygenase (5-LOX) and cyclooxygenases (constitutive COX-1 and inducible COX-2). It is believed that dual inhibition of 5-LOX and COXs may have a higher beneficial impact in the treatment of inflammatory disorders rather than the inhibition of each enzyme. With this demand for new dual-acting anti-inflammatory agents, a range of 2,3-diarylxanthones were tested through their ability to interact in the AA metabolism. In vitro anti-inflammatory activity was evaluated through the inhibition of 5-LOX-catalyzed leukotriene B4 (LTB4) formation in human neutrophils and inhibition of COX-1- and COX-2-catalyzed prostaglandin E2 (PGE2) formation in human whole blood. The results showed that some of the studied arylxanthones were able to prevent LTB4 production in human neutrophils, in a concentration-dependent manner. The xanthone with a 2-catechol was the most active one (IC50 ∼ 9 μM). The more effective arylxanthones in preventing COX-1-catalyzed PGE2 production presented IC50 values from 1 to 7 μM, exhibiting a structural feature with at least one non-substituted aryl group. All the studied arylxanthones were ineffective to prevent the formation of PGE2 catalyzed by COX-2, up to the maximum concentration of 100 μM. The ability of the tested 2,3-diarylxanthones to interact with both 5-LOX and COX-1 pathways constitutes an important step in the research of novel dual-acting anti-inflammatory drugs.application/pdfpt_PT2,3-Diarylxanthones as potential inhibitors of Arachidonic acid metabolic pathwaysPersonalSantos, Clementina M.M.DSpacehttp://dspace.org/items/64f662b6-775d-4ea0-bfd7-f527af0ac1a0DSpacehttp://dspace.org/items/64f662b6-775d-4ea0-bfd7-f527af0ac1a0SantosClementina M.M.Ciência IDhttps://www.ciencia-id.pt9018-DB9C-C590ORCIDhttp://orcid.org0000-0003-4380-7990Scopus Author IDhttps://www.scopus.com7201458663Ribeiro, DanielaSilva, ArturFernandes, EduardaHostingInstitutionOrganizationalBiblioteca Digital do IPBe-mailmailto:dspace@ipb.ptdspace@ipb.ptDOIIsPartOf10.1007/s10753-017-0540-62017-09-22T10:59:41Z20172017-01-01T00:00:00ZHandlehttp://hdl.handle.net/10198/14519http://purl.org/coar/access_right/c_abf2open accessXanthones5-LOXCOX-1COX-2Human neutrophilsHuman whole-blood assays1259543 bytesliteraturehttp://purl.org/coar/resource_type/c_6501journal articlehttp://purl.org/coar/access_right/c_abf2application/pdffulltexthttps://bibliotecadigital.ipb.pt/bitstreams/292cb167-ce95-4d66-b8d0-6027e5a0b9d8/downloadInflammation403956964
spellingShingle 2,3-Diarylxanthones as potential inhibitors of Arachidonic acid metabolic pathways
Santos, Clementina M.M.
Xanthones
5-LOX
COX-1
COX-2
Human neutrophils
Human whole-blood assays
status SINGLETON
subject.fl_str_mv Xanthones
5-LOX
COX-1
COX-2
Human neutrophils
Human whole-blood assays
title 2,3-Diarylxanthones as potential inhibitors of Arachidonic acid metabolic pathways
title_full 2,3-Diarylxanthones as potential inhibitors of Arachidonic acid metabolic pathways
title_fullStr 2,3-Diarylxanthones as potential inhibitors of Arachidonic acid metabolic pathways
title_full_unstemmed 2,3-Diarylxanthones as potential inhibitors of Arachidonic acid metabolic pathways
title_short 2,3-Diarylxanthones as potential inhibitors of Arachidonic acid metabolic pathways
title_sort 2,3-Diarylxanthones as potential inhibitors of Arachidonic acid metabolic pathways
topic Xanthones
5-LOX
COX-1
COX-2
Human neutrophils
Human whole-blood assays
topic_facet Xanthones
5-LOX
COX-1
COX-2
Human neutrophils
Human whole-blood assays
url http://hdl.handle.net/10198/14519
visible 1