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Enhanced role of adenosine A2A receptors in the modulation of LTP in the rat hippocampus upon ageing

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Resumo:Adenosine neuromodulation depends on a balanced activation of inhibitory A1 (A1R) and facilitatory A2A receptors (A2AR). Both A1R and A2AR modulate hippocampal glutamate release and NMDA-dependent long-term potentiation (LTP) but ageing affects the density of both A1R and A2AR. We tested the effects of selective A1R and A2AR antagonists in the modulation of synaptic transmission and plasticity in rat hippocampal slices from three age groups (young adults, 2–3 month; middle-aged adults, 6–8 months; aged, 18–20 months). The selective A2AR antagonist SCH58261 (50 nm) attenuated LTP in all age groups, with a larger effect in aged ()63 ± 7%) than in middle-aged adults ()36 ± 9%) or young adult rats ()36 ± 9%). In contrast, the selective A1R antagonist DPCPX (50 nm) increased LTP magnitude in young adult rats (+42 ± 6%), but failed to affect LTP magnitude in the other age groups. Finally, in the continuous presence of DPCPX, SCH58261 caused a significantly larger inhibition of LTP amplitude in aged ()71 ± 45%) than middle-aged ()28 ± 9%) or young rats ()11 ± 2%). Accordingly, aged rats displayed an increased expression of A2AR mRNA in the hippocampus and a higher number of glutamatergic nerve terminals equipped with A2AR in aged (67 ± 6%) compared with middle-aged (34 ± 7%) and young rats (25 ± 5%). The results show an enhanced A2AR-mediated modulation of LTP in aged rats, in accordance with the age-associated increased expression and density of A2AR in glutamatergic terminals. This age-associated gain of function of A2AR modulating synaptic plasticity may underlie the ability of A2AR antagonists to prevent memory dysfunction in aged animals.
Autores principais:Costenla, Ana R.
Outros Autores:Diógenes, Maria J.; Canas, Paula M.; Rodrigues, Ricardo J.; Nogueira, Célia; Maroco, João; Agostinho, Paula M.; Ribeiro, Joaquim A.; Cunha, Rodrigo A.; Mendonça, Alexandre de
Assunto:A1 receptors A2A receptors Adenosine Ageing Hippocampus Synaptic plasticity
Ano:2011
País:Portugal
Tipo de documento:artigo
Tipo de acesso:acesso aberto
Instituição associada:Ispa-Instituto Universitário
Idioma:inglês
Origem:Repositório do Ispa - Instituto Universitário
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author Costenla, Ana R.
author2 Diógenes, Maria J.
Canas, Paula M.
Rodrigues, Ricardo J.
Nogueira, Célia
Maroco, João
Agostinho, Paula M.
Ribeiro, Joaquim A.
Cunha, Rodrigo A.
Mendonça, Alexandre de
author2_role author
author
author
author
author
author
author
author
author
author_facet Costenla, Ana R.
Diógenes, Maria J.
Canas, Paula M.
Rodrigues, Ricardo J.
Nogueira, Célia
Maroco, João
Agostinho, Paula M.
Ribeiro, Joaquim A.
Cunha, Rodrigo A.
Mendonça, Alexandre de
author_role author
contributor_name_str_mv Repositório do ISPA
country_str PT
creators_json_txt [{\"Person.name\":\"Costenla, Ana R.\"},{\"Person.name\":\"Diógenes, Maria J.\"},{\"Person.name\":\"Canas, Paula M.\"},{\"Person.name\":\"Rodrigues, Ricardo J.\"},{\"Person.name\":\"Nogueira, Célia\"},{\"Person.name\":\"Maroco, João\"},{\"Person.name\":\"Agostinho, Paula M.\"},{\"Person.name\":\"Ribeiro, Joaquim A.\"},{\"Person.name\":\"Cunha, Rodrigo A.\"},{\"Person.name\":\"Mendonça, Alexandre de\"}]
datacite.contributors.contributor.contributorName.fl_str_mv Repositório do ISPA
datacite.creators.creator.creatorName.fl_str_mv Costenla, Ana R.
Diógenes, Maria J.
Canas, Paula M.
Rodrigues, Ricardo J.
Nogueira, Célia
Maroco, João
Agostinho, Paula M.
Ribeiro, Joaquim A.
Cunha, Rodrigo A.
Mendonça, Alexandre de
datacite.date.Accepted.fl_str_mv 2011-01-01T00:00:00Z
datacite.date.available.fl_str_mv 2012-02-16T20:58:32Z
datacite.date.embargoed.fl_str_mv 2012-02-16T20:58:32Z
datacite.rights.fl_str_mv http://purl.org/coar/access_right/c_abf2
datacite.subjects.subject.fl_str_mv A1 receptors
A2A receptors
Adenosine
Ageing
Hippocampus
Synaptic plasticity
datacite.titles.title.fl_str_mv Enhanced role of adenosine A2A receptors in the modulation of LTP in the rat hippocampus upon ageing
dc.contributor.none.fl_str_mv Repositório do ISPA
dc.creator.none.fl_str_mv Costenla, Ana R.
Diógenes, Maria J.
Canas, Paula M.
Rodrigues, Ricardo J.
Nogueira, Célia
Maroco, João
Agostinho, Paula M.
Ribeiro, Joaquim A.
Cunha, Rodrigo A.
Mendonça, Alexandre de
dc.date.Accepted.fl_str_mv 2011-01-01T00:00:00Z
dc.date.available.fl_str_mv 2012-02-16T20:58:32Z
dc.date.embargoed.fl_str_mv 2012-02-16T20:58:32Z
dc.format.none.fl_str_mv application/pdf
dc.identifier.none.fl_str_mv http://hdl.handle.net/10400.12/1237
dc.language.none.fl_str_mv eng
dc.publisher.none.fl_str_mv Blackwell Publishing
dc.rights.none.fl_str_mv http://purl.org/coar/access_right/c_abf2
dc.subject.none.fl_str_mv A1 receptors
A2A receptors
Adenosine
Ageing
Hippocampus
Synaptic plasticity
dc.title.fl_str_mv Enhanced role of adenosine A2A receptors in the modulation of LTP in the rat hippocampus upon ageing
dc.type.none.fl_str_mv http://purl.org/coar/resource_type/c_6501
description Adenosine neuromodulation depends on a balanced activation of inhibitory A1 (A1R) and facilitatory A2A receptors (A2AR). Both A1R and A2AR modulate hippocampal glutamate release and NMDA-dependent long-term potentiation (LTP) but ageing affects the density of both A1R and A2AR. We tested the effects of selective A1R and A2AR antagonists in the modulation of synaptic transmission and plasticity in rat hippocampal slices from three age groups (young adults, 2–3 month; middle-aged adults, 6–8 months; aged, 18–20 months). The selective A2AR antagonist SCH58261 (50 nm) attenuated LTP in all age groups, with a larger effect in aged ()63 ± 7%) than in middle-aged adults ()36 ± 9%) or young adult rats ()36 ± 9%). In contrast, the selective A1R antagonist DPCPX (50 nm) increased LTP magnitude in young adult rats (+42 ± 6%), but failed to affect LTP magnitude in the other age groups. Finally, in the continuous presence of DPCPX, SCH58261 caused a significantly larger inhibition of LTP amplitude in aged ()71 ± 45%) than middle-aged ()28 ± 9%) or young rats ()11 ± 2%). Accordingly, aged rats displayed an increased expression of A2AR mRNA in the hippocampus and a higher number of glutamatergic nerve terminals equipped with A2AR in aged (67 ± 6%) compared with middle-aged (34 ± 7%) and young rats (25 ± 5%). The results show an enhanced A2AR-mediated modulation of LTP in aged rats, in accordance with the age-associated increased expression and density of A2AR in glutamatergic terminals. This age-associated gain of function of A2AR modulating synaptic plasticity may underlie the ability of A2AR antagonists to prevent memory dysfunction in aged animals.
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eu_rights_str_mv openAccess
format article
fulltext.url.fl_str_mv https://repositorio.ispa.pt/bitstreams/3944ecbb-91e8-4da2-90bd-ab44d138716d/download
id ispa_bbd0334d2bb930d678af6880425eee35
identifier.url.fl_str_mv http://hdl.handle.net/10400.12/1237
instacron_str ispa
institution Ispa-Instituto Universitário
instname_str Ispa-Instituto Universitário
language eng
network_acronym_str ispa
network_name_str Repositório do Ispa - Instituto Universitário
oai_identifier_str oai:repositorio.ispa.pt:10400.12/1237
organization_str_mv urn:organizationAcronym:ispa
person_str_mv Costenla, Ana R.
Diógenes, Maria J.
Canas, Paula M.
Rodrigues, Ricardo J.
Nogueira, Célia
Maroco, João
Agostinho, Paula M.
Ribeiro, Joaquim A.
Cunha, Rodrigo A.
Mendonça, Alexandre de
publishDate 2011
publisher.none.fl_str_mv Blackwell Publishing
reponame_str Repositório do Ispa - Instituto Universitário
repository_id_str urn:repositoryAcronym:ispa
service_str_mv urn:repositoryAcronym:ispa
spelling engBlackwell PublishingporAdenosine neuromodulation depends on a balanced activation of inhibitory A1 (A1R) and facilitatory A2A receptors (A2AR). Both A1R and A2AR modulate hippocampal glutamate release and NMDA-dependent long-term potentiation (LTP) but ageing affects the density of both A1R and A2AR. We tested the effects of selective A1R and A2AR antagonists in the modulation of synaptic transmission and plasticity in rat hippocampal slices from three age groups (young adults, 2–3 month; middle-aged adults, 6–8 months; aged, 18–20 months). The selective A2AR antagonist SCH58261 (50 nm) attenuated LTP in all age groups, with a larger effect in aged ()63 ± 7%) than in middle-aged adults ()36 ± 9%) or young adult rats ()36 ± 9%). In contrast, the selective A1R antagonist DPCPX (50 nm) increased LTP magnitude in young adult rats (+42 ± 6%), but failed to affect LTP magnitude in the other age groups. Finally, in the continuous presence of DPCPX, SCH58261 caused a significantly larger inhibition of LTP amplitude in aged ()71 ± 45%) than middle-aged ()28 ± 9%) or young rats ()11 ± 2%). Accordingly, aged rats displayed an increased expression of A2AR mRNA in the hippocampus and a higher number of glutamatergic nerve terminals equipped with A2AR in aged (67 ± 6%) compared with middle-aged (34 ± 7%) and young rats (25 ± 5%). The results show an enhanced A2AR-mediated modulation of LTP in aged rats, in accordance with the age-associated increased expression and density of A2AR in glutamatergic terminals. This age-associated gain of function of A2AR modulating synaptic plasticity may underlie the ability of A2AR antagonists to prevent memory dysfunction in aged animals.application/pdfporEnhanced role of adenosine A2A receptors in the modulation of LTP in the rat hippocampus upon ageingCostenla, Ana R.Diógenes, Maria J.Canas, Paula M.Rodrigues, Ricardo J.Nogueira, CéliaMaroco, JoãoAgostinho, Paula M.Ribeiro, Joaquim A.Cunha, Rodrigo A.Mendonça, Alexandre deHostingInstitutionOrganizationalRepositório do ISPAe-mailmailto:repositorio@ispa.ptrepositorio@ispa.ptISSNIsPartOf1460-95682012-02-16T20:58:32Z20112011-01-01T00:00:00ZHandlehttp://hdl.handle.net/10400.12/1237http://purl.org/coar/access_right/c_abf2open accessA1 receptorsA2A receptorsAdenosineAgeingHippocampusSynaptic plasticity484794 bytesliteraturehttp://purl.org/coar/resource_type/c_6501journal articlehttp://purl.org/coar/access_right/c_abf2application/pdffulltexthttps://repositorio.ispa.pt/bitstreams/3944ecbb-91e8-4da2-90bd-ab44d138716d/downloadEuropean Journal of Neuroscience1221Zurich
spellingShingle Enhanced role of adenosine A2A receptors in the modulation of LTP in the rat hippocampus upon ageing
Costenla, Ana R.
A1 receptors
A2A receptors
Adenosine
Ageing
Hippocampus
Synaptic plasticity
status SINGLETON
subject.fl_str_mv A1 receptors
A2A receptors
Adenosine
Ageing
Hippocampus
Synaptic plasticity
title Enhanced role of adenosine A2A receptors in the modulation of LTP in the rat hippocampus upon ageing
title_full Enhanced role of adenosine A2A receptors in the modulation of LTP in the rat hippocampus upon ageing
title_fullStr Enhanced role of adenosine A2A receptors in the modulation of LTP in the rat hippocampus upon ageing
title_full_unstemmed Enhanced role of adenosine A2A receptors in the modulation of LTP in the rat hippocampus upon ageing
title_short Enhanced role of adenosine A2A receptors in the modulation of LTP in the rat hippocampus upon ageing
title_sort Enhanced role of adenosine A2A receptors in the modulation of LTP in the rat hippocampus upon ageing
topic A1 receptors
A2A receptors
Adenosine
Ageing
Hippocampus
Synaptic plasticity
topic_facet A1 receptors
A2A receptors
Adenosine
Ageing
Hippocampus
Synaptic plasticity
url http://hdl.handle.net/10400.12/1237
visible 1