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Polyglycine expansions in eRF3/GSPT1 are associated with gastric cancer susceptibility

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Resumo:Gastric cancer remains a major cause of death in the developed countries, and a large percentage is still genetically unexplained. Because of their major role in cell survival, mutations in translation factors and altered expression of these genes have been associated with cancer development. Apart from its role in translation termination, the eukaryotic translation release factor 3 (eRF3) is involved in several critical cellular processes, such as cell cycle regulation, cytoskeleton organization, and apoptosis. The aim of this study was to evaluate eRF3/GSPT1 gene as a potential genetic susceptibility associated locus for gastric cancer, analyzing a stable GGC expansion in exon 1 encoding a polyglycine tract in the N-terminal domain of the protein. DNA was obtained from 139 patients with gastric cancer and from 100 individuals of a healthy control population. The GGC expansion was amplified by PCR and the number of repeats determined by genotyping in an automatic sequencer. There are five known alleles encoding from 8 to 12 glycines. The most common allele encodes 10 glycines. The 12-Gly allele was detected exclusively in the cancer patients (allelic frequency = 5%). Regardless of the genotype, patients with the 12-Gly allele had a 20-fold increased risk for gastric cancer. We also detected a single-base alteration in the gene (G274T) although no correlation with cancer development has been found. Thus, our results show that the GGC expansion may have a potential role in regulating eRF3/GSPT1 expression and/or changing the protein function that can lead to gastric cancer development.
Autores principais:Brito, Miguel
Outros Autores:Malta-Vacas, Joana; Carmona, Bruno; Aires, C.; Costa, P.; Martins, A. P.; Ramos, S.; Conde, A. R.; Monteiro, C.
Assunto:Adenocarcinoma Adenocarcinoma, Mucinous Amino acid sequence DNA Disease susceptibility Exons Molecular sequence data Peptide termination factors Peptides Sequence homology, Amino acid Stomach neoplasms Trinucleotide repeat expansion
Ano:2005
País:Portugal
Tipo de documento:artigo
Tipo de acesso:acesso aberto
Instituição associada:Instituto Politécnico de Lisboa
Idioma:inglês
Origem:Repositório Científico do Instituto Politécnico de Lisboa
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author Brito, Miguel
author2 Malta-Vacas, Joana
Carmona, Bruno
Aires, C.
Costa, P.
Martins, A. P.
Ramos, S.
Conde, A. R.
Monteiro, C.
author2_role author
author
author
author
author
author
author
author
author_facet Brito, Miguel
Malta-Vacas, Joana
Carmona, Bruno
Aires, C.
Costa, P.
Martins, A. P.
Ramos, S.
Conde, A. R.
Monteiro, C.
author_role author
contributor_name_str_mv RCIPL
country_str PT
creators_json_txt [{\"Person.name\":\"Brito, Miguel\",\"Person.identifier.orcid\":\"0000-0001-6394-658X\"},{\"Person.name\":\"Malta-Vacas, Joana\"},{\"Person.name\":\"Carmona, Bruno\"},{\"Person.name\":\"Aires, C.\"},{\"Person.name\":\"Costa, P.\"},{\"Person.name\":\"Martins, A. P.\"},{\"Person.name\":\"Ramos, S.\"},{\"Person.name\":\"Conde, A. R.\"},{\"Person.name\":\"Monteiro, C.\"}]
datacite.contributors.contributor.contributorName.fl_str_mv RCIPL
datacite.creators.creator.creatorName.fl_str_mv Brito, Miguel
Malta-Vacas, Joana
Carmona, Bruno
Aires, C.
Costa, P.
Martins, A. P.
Ramos, S.
Conde, A. R.
Monteiro, C.
datacite.date.Accepted.fl_str_mv 2005-12-01T00:00:00Z
datacite.date.available.fl_str_mv 2018-06-11T16:56:47Z
datacite.date.embargoed.fl_str_mv 2018-06-11T16:56:47Z
datacite.rights.fl_str_mv http://purl.org/coar/access_right/c_abf2
datacite.subjects.subject.fl_str_mv Adenocarcinoma
Adenocarcinoma, Mucinous
Amino acid sequence
DNA
Disease susceptibility
Exons
Molecular sequence data
Peptide termination factors
Peptides
Sequence homology, Amino acid
Stomach neoplasms
Trinucleotide repeat expansion
datacite.titles.title.fl_str_mv Polyglycine expansions in eRF3/GSPT1 are associated with gastric cancer susceptibility
dc.contributor.none.fl_str_mv RCIPL
dc.creator.none.fl_str_mv Brito, Miguel
Malta-Vacas, Joana
Carmona, Bruno
Aires, C.
Costa, P.
Martins, A. P.
Ramos, S.
Conde, A. R.
Monteiro, C.
dc.date.Accepted.fl_str_mv 2005-12-01T00:00:00Z
dc.date.available.fl_str_mv 2018-06-11T16:56:47Z
dc.date.embargoed.fl_str_mv 2018-06-11T16:56:47Z
dc.format.none.fl_str_mv application/pdf
dc.identifier.none.fl_str_mv http://hdl.handle.net/10400.21/8620
dc.language.none.fl_str_mv eng
dc.publisher.none.fl_str_mv Oxford University Press
dc.rights.cclincense.fl_str_mv http://creativecommons.org/licenses/by-nc-nd/4.0/
dc.rights.none.fl_str_mv http://purl.org/coar/access_right/c_abf2
dc.subject.none.fl_str_mv Adenocarcinoma
Adenocarcinoma, Mucinous
Amino acid sequence
DNA
Disease susceptibility
Exons
Molecular sequence data
Peptide termination factors
Peptides
Sequence homology, Amino acid
Stomach neoplasms
Trinucleotide repeat expansion
dc.title.fl_str_mv Polyglycine expansions in eRF3/GSPT1 are associated with gastric cancer susceptibility
dc.type.none.fl_str_mv http://purl.org/coar/resource_type/c_6501
description Gastric cancer remains a major cause of death in the developed countries, and a large percentage is still genetically unexplained. Because of their major role in cell survival, mutations in translation factors and altered expression of these genes have been associated with cancer development. Apart from its role in translation termination, the eukaryotic translation release factor 3 (eRF3) is involved in several critical cellular processes, such as cell cycle regulation, cytoskeleton organization, and apoptosis. The aim of this study was to evaluate eRF3/GSPT1 gene as a potential genetic susceptibility associated locus for gastric cancer, analyzing a stable GGC expansion in exon 1 encoding a polyglycine tract in the N-terminal domain of the protein. DNA was obtained from 139 patients with gastric cancer and from 100 individuals of a healthy control population. The GGC expansion was amplified by PCR and the number of repeats determined by genotyping in an automatic sequencer. There are five known alleles encoding from 8 to 12 glycines. The most common allele encodes 10 glycines. The 12-Gly allele was detected exclusively in the cancer patients (allelic frequency = 5%). Regardless of the genotype, patients with the 12-Gly allele had a 20-fold increased risk for gastric cancer. We also detected a single-base alteration in the gene (G274T) although no correlation with cancer development has been found. Thus, our results show that the GGC expansion may have a potential role in regulating eRF3/GSPT1 expression and/or changing the protein function that can lead to gastric cancer development.
dirty 0
eu_rights_str_mv openAccess
format article
fulltext.url.fl_str_mv https://repositorio.ipl.pt/bitstreams/b2a91b36-2c88-4d45-94bd-e1f7d9dede15/download
id ripl_744aef7acf95abcbbccdcd1f11b10e2e
identifier.url.fl_str_mv http://hdl.handle.net/10400.21/8620
instacron_str ipl
institution Instituto Politécnico de Lisboa
instname_str Instituto Politécnico de Lisboa
language eng
network_acronym_str ripl
network_name_str Repositório Científico do Instituto Politécnico de Lisboa
oai_identifier_str oai:repositorio.ipl.pt:10400.21/8620
organization_str_mv urn:organizationAcronym:ipl
person_str_mv Brito, Miguel
Brito, Miguel
https://www.ciencia-id.pt/231F-F341-7E93
231F-F341-7E93
http://orcid.org/0000-0001-6394-658X
0000-0001-6394-658X
Malta-Vacas, Joana
Carmona, Bruno
Aires, C.
Costa, P.
Martins, A. P.
Ramos, S.
Conde, A. R.
Monteiro, C.
publishDate 2005
publisher.none.fl_str_mv Oxford University Press
reponame_str Repositório Científico do Instituto Politécnico de Lisboa
repository_id_str urn:repositoryAcronym:ripl
service_str_mv urn:repositoryAcronym:ripl
spelling engOxford University Presspt_PTGastric cancer remains a major cause of death in the developed countries, and a large percentage is still genetically unexplained. Because of their major role in cell survival, mutations in translation factors and altered expression of these genes have been associated with cancer development. Apart from its role in translation termination, the eukaryotic translation release factor 3 (eRF3) is involved in several critical cellular processes, such as cell cycle regulation, cytoskeleton organization, and apoptosis. The aim of this study was to evaluate eRF3/GSPT1 gene as a potential genetic susceptibility associated locus for gastric cancer, analyzing a stable GGC expansion in exon 1 encoding a polyglycine tract in the N-terminal domain of the protein. DNA was obtained from 139 patients with gastric cancer and from 100 individuals of a healthy control population. The GGC expansion was amplified by PCR and the number of repeats determined by genotyping in an automatic sequencer. There are five known alleles encoding from 8 to 12 glycines. The most common allele encodes 10 glycines. The 12-Gly allele was detected exclusively in the cancer patients (allelic frequency = 5%). Regardless of the genotype, patients with the 12-Gly allele had a 20-fold increased risk for gastric cancer. We also detected a single-base alteration in the gene (G274T) although no correlation with cancer development has been found. Thus, our results show that the GGC expansion may have a potential role in regulating eRF3/GSPT1 expression and/or changing the protein function that can lead to gastric cancer development.application/pdfpt_PTPolyglycine expansions in eRF3/GSPT1 are associated with gastric cancer susceptibilityPersonalBrito, MiguelDSpacehttp://dspace.org/items/4252d8e0-800c-4d67-8b13-0b711d860669DSpacehttp://dspace.org/items/4252d8e0-800c-4d67-8b13-0b711d860669BritoMiguelCiência IDhttps://www.ciencia-id.pt231F-F341-7E93ORCIDhttp://orcid.org0000-0001-6394-658XResearcher IDhttps://www.researcherid.comA-7970-2016Scopus Author IDhttps://www.scopus.com35224551000Scopus Author IDhttps://www.scopus.com57200288349Malta-Vacas, JoanaCarmona, BrunoAires, C.Costa, P.Martins, A. P.Ramos, S.Conde, A. R.Monteiro, C.HostingInstitutionOrganizationalRCIPLe-mailmailto:rcaap@sp.ipl.ptrcaap@sp.ipl.ptDOIIsPartOf10.1093/carcin/bgi1682018-06-11T16:56:47Z2005-122005-12-01T00:00:00ZHandlehttp://hdl.handle.net/10400.21/8620http://purl.org/coar/access_right/c_abf2open accessAdenocarcinomaAdenocarcinoma, MucinousAmino acid sequenceDNADisease susceptibilityExonsMolecular sequence dataPeptide termination factorsPeptidesSequence homology, Amino acidStomach neoplasmsTrinucleotide repeat expansion123273 bytesliteraturehttp://purl.org/coar/resource_type/c_6501journal article2005-12http://creativecommons.org/licenses/by-nc-nd/4.0/http://purl.org/coar/access_right/c_abf2application/pdffulltexthttps://repositorio.ipl.pt/bitstreams/b2a91b36-2c88-4d45-94bd-e1f7d9dede15/downloadCarcinogenesis261220462049
spellingShingle Polyglycine expansions in eRF3/GSPT1 are associated with gastric cancer susceptibility
Brito, Miguel
Adenocarcinoma
Adenocarcinoma, Mucinous
Amino acid sequence
DNA
Disease susceptibility
Exons
Molecular sequence data
Peptide termination factors
Peptides
Sequence homology, Amino acid
Stomach neoplasms
Trinucleotide repeat expansion
status SINGLETON
subject.fl_str_mv Adenocarcinoma
Adenocarcinoma, Mucinous
Amino acid sequence
DNA
Disease susceptibility
Exons
Molecular sequence data
Peptide termination factors
Peptides
Sequence homology, Amino acid
Stomach neoplasms
Trinucleotide repeat expansion
title Polyglycine expansions in eRF3/GSPT1 are associated with gastric cancer susceptibility
title_full Polyglycine expansions in eRF3/GSPT1 are associated with gastric cancer susceptibility
title_fullStr Polyglycine expansions in eRF3/GSPT1 are associated with gastric cancer susceptibility
title_full_unstemmed Polyglycine expansions in eRF3/GSPT1 are associated with gastric cancer susceptibility
title_short Polyglycine expansions in eRF3/GSPT1 are associated with gastric cancer susceptibility
title_sort Polyglycine expansions in eRF3/GSPT1 are associated with gastric cancer susceptibility
topic Adenocarcinoma
Adenocarcinoma, Mucinous
Amino acid sequence
DNA
Disease susceptibility
Exons
Molecular sequence data
Peptide termination factors
Peptides
Sequence homology, Amino acid
Stomach neoplasms
Trinucleotide repeat expansion
topic_facet Adenocarcinoma
Adenocarcinoma, Mucinous
Amino acid sequence
DNA
Disease susceptibility
Exons
Molecular sequence data
Peptide termination factors
Peptides
Sequence homology, Amino acid
Stomach neoplasms
Trinucleotide repeat expansion
url http://hdl.handle.net/10400.21/8620
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