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Brain barriers and the acute-phase response

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Resumo:[Excerpt] Recent findings on the brain response to peripheral inflammation have broadened our knowledge on the acute phase response. Notably, such advances may lead to the characterization of acute phase proteins in body fluids other than the blood. Among these, and of particular interest, is the cerebrospinal fluid (CSF) that fills the brain ventricles, the subarachnoid space and the spinal cord. Acute phase response is a term used to describe, mostly, the liver response to an inflammatory insult, a process that, among other effects, ultimately results in changes in the plasma concentrations of various proteins. The triggers of liver response include the inflammatory insult and the molecules produced by multiple cell types, from inflammatory cells (e.g. neutrophils and macrophages), to endothelial and liver cells. As a consequence, the liver synthesizes a number of so-termed acute phase proteins that are classified, depending on whether their expression is increased or decreased, as positive or negative acute phase response proteins, respectively. Among the positive acute phase proteins are binding proteins, opsonins (e.g. C-reactive protein) and iron-related proteins [e.g. hepcidin (HAMP), ceruloplasmin, lipocalin 2 (LCN2) and ferritin]. The negative acute phase proteins also include binding proteins (e.g. albumin, transthyretin, retinol-binding protein), as well as iron-related proteins (e.g. transferrin). Nonetheless, acute phase response is not a process restricted to the liver. Throughout the years, acute phase proteins expression has also been found altered at the barriers of the brain. Most of these findings originated from work on the brain parenchyma response to inflammation (Quan et al., 1998; Nadeau & Rivest, 1999); however, surprisingly, only recently were the barriers of the brain specifically considered in the overall response to peripheral inflammation. In this chapter we will address the contribution of the barriers of the brain in the acute-phase response to peripheral inflammatory stimuli. [...]
Autores principais:Marques, Fernanda
Outros Autores:Correia-Neves, M; Sousa, João Carlos; Sousa, Nuno; Palha, Joana Almeida
Assunto:Ciências Médicas::Medicina Básica
Ano:2011
País:Portugal
Tipo de documento:capítulo de livro
Tipo de acesso:acesso restrito
Instituição associada:Universidade do Minho
Idioma:inglês
Origem:RepositóriUM - Universidade do Minho
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author Marques, Fernanda
author2 Correia-Neves, M
Sousa, João Carlos
Sousa, Nuno
Palha, Joana Almeida
author2_role author
author
author
author
author_facet Marques, Fernanda
Correia-Neves, M
Sousa, João Carlos
Sousa, Nuno
Palha, Joana Almeida
author_role author
contributor_name_str_mv RepositóriUM - Universidade do Minho
country_str PT
creators_json_txt [{\"Person.name\":\"Marques, Fernanda\"},{\"Person.name\":\"Correia-Neves, M\"},{\"Person.name\":\"Sousa, João Carlos\"},{\"Person.name\":\"Sousa, Nuno\"},{\"Person.name\":\"Palha, Joana Almeida\"}]
datacite.contributors.contributor.contributorName.fl_str_mv RepositóriUM - Universidade do Minho
datacite.creators.creator.creatorName.fl_str_mv Marques, Fernanda
Correia-Neves, M
Sousa, João Carlos
Sousa, Nuno
Palha, Joana Almeida
datacite.date.Accepted.fl_str_mv 2011-10-01T00:00:00Z
datacite.date.embargoed.fl_str_mv 10000-01-01T00:00:00Z
datacite.rights.fl_str_mv http://purl.org/coar/access_right/c_16ec
datacite.subjects.subject.fl_str_mv Ciências Médicas::Medicina Básica
datacite.titles.title.fl_str_mv Brain barriers and the acute-phase response
dc.contributor.none.fl_str_mv RepositóriUM - Universidade do Minho
dc.creator.none.fl_str_mv Marques, Fernanda
Correia-Neves, M
Sousa, João Carlos
Sousa, Nuno
Palha, Joana Almeida
dc.date.Accepted.fl_str_mv 2011-10-01T00:00:00Z
dc.date.embargoed.fl_str_mv 10000-01-01T00:00:00Z
dc.format.none.fl_str_mv application/pdf
dc.identifier.none.fl_str_mv https://hdl.handle.net/1822/67447
dc.language.none.fl_str_mv eng
dc.publisher.none.fl_str_mv IntechOpen
dc.rights.none.fl_str_mv http://purl.org/coar/access_right/c_16ec
dc.subject.none.fl_str_mv Ciências Médicas::Medicina Básica
dc.title.fl_str_mv Brain barriers and the acute-phase response
dc.type.none.fl_str_mv http://purl.org/coar/resource_type/c_3248
description [Excerpt] Recent findings on the brain response to peripheral inflammation have broadened our knowledge on the acute phase response. Notably, such advances may lead to the characterization of acute phase proteins in body fluids other than the blood. Among these, and of particular interest, is the cerebrospinal fluid (CSF) that fills the brain ventricles, the subarachnoid space and the spinal cord. Acute phase response is a term used to describe, mostly, the liver response to an inflammatory insult, a process that, among other effects, ultimately results in changes in the plasma concentrations of various proteins. The triggers of liver response include the inflammatory insult and the molecules produced by multiple cell types, from inflammatory cells (e.g. neutrophils and macrophages), to endothelial and liver cells. As a consequence, the liver synthesizes a number of so-termed acute phase proteins that are classified, depending on whether their expression is increased or decreased, as positive or negative acute phase response proteins, respectively. Among the positive acute phase proteins are binding proteins, opsonins (e.g. C-reactive protein) and iron-related proteins [e.g. hepcidin (HAMP), ceruloplasmin, lipocalin 2 (LCN2) and ferritin]. The negative acute phase proteins also include binding proteins (e.g. albumin, transthyretin, retinol-binding protein), as well as iron-related proteins (e.g. transferrin). Nonetheless, acute phase response is not a process restricted to the liver. Throughout the years, acute phase proteins expression has also been found altered at the barriers of the brain. Most of these findings originated from work on the brain parenchyma response to inflammation (Quan et al., 1998; Nadeau & Rivest, 1999); however, surprisingly, only recently were the barriers of the brain specifically considered in the overall response to peripheral inflammation. In this chapter we will address the contribution of the barriers of the brain in the acute-phase response to peripheral inflammatory stimuli. [...]
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person_str_mv Marques, Fernanda
Correia-Neves, M
Sousa, João Carlos
Sousa, Nuno
Palha, Joana Almeida
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spelling engIntechOpen[Excerpt] Recent findings on the brain response to peripheral inflammation have broadened our knowledge on the acute phase response. Notably, such advances may lead to the characterization of acute phase proteins in body fluids other than the blood. Among these, and of particular interest, is the cerebrospinal fluid (CSF) that fills the brain ventricles, the subarachnoid space and the spinal cord. Acute phase response is a term used to describe, mostly, the liver response to an inflammatory insult, a process that, among other effects, ultimately results in changes in the plasma concentrations of various proteins. The triggers of liver response include the inflammatory insult and the molecules produced by multiple cell types, from inflammatory cells (e.g. neutrophils and macrophages), to endothelial and liver cells. As a consequence, the liver synthesizes a number of so-termed acute phase proteins that are classified, depending on whether their expression is increased or decreased, as positive or negative acute phase response proteins, respectively. Among the positive acute phase proteins are binding proteins, opsonins (e.g. C-reactive protein) and iron-related proteins [e.g. hepcidin (HAMP), ceruloplasmin, lipocalin 2 (LCN2) and ferritin]. The negative acute phase proteins also include binding proteins (e.g. albumin, transthyretin, retinol-binding protein), as well as iron-related proteins (e.g. transferrin). Nonetheless, acute phase response is not a process restricted to the liver. Throughout the years, acute phase proteins expression has also been found altered at the barriers of the brain. Most of these findings originated from work on the brain parenchyma response to inflammation (Quan et al., 1998; Nadeau & Rivest, 1999); however, surprisingly, only recently were the barriers of the brain specifically considered in the overall response to peripheral inflammation. In this chapter we will address the contribution of the barriers of the brain in the acute-phase response to peripheral inflammatory stimuli. [...]application/pdfporBrain barriers and the acute-phase responseMarques, FernandaCorreia-Neves, MSousa, João CarlosSousa, NunoPalha, Joana AlmeidaHostingInstitutionOrganizationalRepositóriUM - Universidade do Minhoe-mailmailto:repositorium@usdb.uminho.ptrepositorium@usdb.uminho.ptISBNIsPartOf978-953-307-252-4DOIIsPartOf10.5772/22780EISBNIsPartOf978-953-51-4424-32011-102011-10-01T00:00:00Z10000-01-01T00:00:00ZHandlehttps://hdl.handle.net/1822/67447http://purl.org/coar/access_right/c_16ecrestricted accesshttp://www.oecd.org/science/inno/38235147.pdfFields of Science and Technology (FOS)Ciências Médicas::Medicina Básica620735 bytesliteraturehttp://purl.org/coar/resource_type/c_3248book parthttp://purl.org/coar/access_right/c_f1cfapplication/pdffulltexthttps://repositorium.uminho.pt/bitstreams/72d9f673-22bb-42c1-b6fe-a18bfe379b91/download
spellingShingle Brain barriers and the acute-phase response
Marques, Fernanda
Ciências Médicas::Medicina Básica
status SINGLETON
subject.other.fl_str_mv Ciências Médicas::Medicina Básica
title Brain barriers and the acute-phase response
title_full Brain barriers and the acute-phase response
title_fullStr Brain barriers and the acute-phase response
title_full_unstemmed Brain barriers and the acute-phase response
title_short Brain barriers and the acute-phase response
title_sort Brain barriers and the acute-phase response
topic Ciências Médicas::Medicina Básica
topic_facet Ciências Médicas::Medicina Básica
url https://hdl.handle.net/1822/67447
visible 1