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Clinical, biochemical and molecular characterization of cystinuria in a cohort of 12 patients

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Summary:Cystinuria is a rare autosomal inherited disorder characterized by impaired transport of cystine and dibasic aminoacids in the proximal renal tubule. Classically, cystinuria is classified as type I (silent heterozygotes) and non-type I (heterozygotes with urinary hyperexcretion of cystine). Molecularly, cystinuria is classified as type A (mutations on SLC3A1 gene) and type B (mutations on SLC7A9 gene). The goal of this study is to provide a comprehensive clinical, biochemical and molecular characterization of a cohort of 12 Portuguese patients affected with cystinuria in order to provide insight into genotype-phenotype correlations. We describe seven type I and five non-type I patients. Regarding the molecular classification, seven patients were type A and five were type B. In SLC3A1 gene, two large genomic rearrangements and 13 sequence variants, including four new variants c.611-2A>C; c.1136+44G>A; c.1597T (p.Y533N); c.*70A>G, were found. One large genomic rearrangement was found in SLC7A9 gene as well as 24 sequence variants including 3 novel variants: c.216C>T (p.C72C), c.1119G>A (p.S373S) and c.*82C>T. In our cohort the most frequent pathogenic mutations were: large rearrangements (33.3% of mutant alleles) and a missense mutation c.1400T>C (p.M467T) (11.1%). This report expands the spectrum of SLC3A1 and SLC7A9 mutations and provides guidance in the clinical implementation of molecular assays in routine genetic counseling of Portuguese patients affected with cystinuria.
Main Authors:Barbosa, Mafalda Fernanda Cabral Santos
Other Authors:Lopes, A.; Mota, C.; Martins, E.; Oliveira, J.; Alves, S.; Bonis, P. de; Mota, M. do Céu; Dias, C.; Santos, P. Rodrigues; Fortuna, A. M.; Quelhas, D.; Lacerda, L.; Biscegliah, L.; Cardoso, M. L.
Subject:Cystinuria MLPA analysis silent mutation SLC3A1 gene SLC7A9 gene
Year:2012
Country:Portugal
Document type:article
Access type:restricted access
Associated institution:Universidade do Minho
Language:English
Origin:RepositóriUM - Universidade do Minho
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author Barbosa, Mafalda Fernanda Cabral Santos
author2 Lopes, A.
Mota, C.
Martins, E.
Oliveira, J.
Alves, S.
Bonis, P. de
Mota, M. do Céu
Dias, C.
Santos, P. Rodrigues
Fortuna, A. M.
Quelhas, D.
Lacerda, L.
Biscegliah, L.
Cardoso, M. L.
author2_role author
author
author
author
author
author
author
author
author
author
author
author
author
author
author_facet Barbosa, Mafalda Fernanda Cabral Santos
Lopes, A.
Mota, C.
Martins, E.
Oliveira, J.
Alves, S.
Bonis, P. de
Mota, M. do Céu
Dias, C.
Santos, P. Rodrigues
Fortuna, A. M.
Quelhas, D.
Lacerda, L.
Biscegliah, L.
Cardoso, M. L.
author_role author
contributor_name_str_mv RepositóriUM - Universidade do Minho
country_str PT
creators_json_txt [{\"Person.name\":\"Barbosa, Mafalda Fernanda Cabral Santos\"},{\"Person.name\":\"Lopes, A.\"},{\"Person.name\":\"Mota, C.\"},{\"Person.name\":\"Martins, E.\"},{\"Person.name\":\"Oliveira, J.\"},{\"Person.name\":\"Alves, S.\"},{\"Person.name\":\"Bonis, P. de\"},{\"Person.name\":\"Mota, M. do Céu\"},{\"Person.name\":\"Dias, C.\"},{\"Person.name\":\"Santos, P. Rodrigues\"},{\"Person.name\":\"Fortuna, A. M.\"},{\"Person.name\":\"Quelhas, D.\"},{\"Person.name\":\"Lacerda, L.\"},{\"Person.name\":\"Biscegliah, L.\"},{\"Person.name\":\"Cardoso, M. L.\"}]
datacite.contributors.contributor.contributorName.fl_str_mv RepositóriUM - Universidade do Minho
datacite.creators.creator.creatorName.fl_str_mv Barbosa, Mafalda Fernanda Cabral Santos
Lopes, A.
Mota, C.
Martins, E.
Oliveira, J.
Alves, S.
Bonis, P. de
Mota, M. do Céu
Dias, C.
Santos, P. Rodrigues
Fortuna, A. M.
Quelhas, D.
Lacerda, L.
Biscegliah, L.
Cardoso, M. L.
datacite.date.Accepted.fl_str_mv 2012-01-01T00:00:00Z
datacite.date.available.fl_str_mv 2015-02-05T12:07:21Z
datacite.date.embargoed.fl_str_mv 2015-02-05T12:07:21Z
datacite.rights.fl_str_mv http://purl.org/coar/access_right/c_16ec
datacite.subjects.subject.fl_str_mv Cystinuria
MLPA analysis
silent mutation
SLC3A1 gene
SLC7A9 gene
datacite.titles.title.fl_str_mv Clinical, biochemical and molecular characterization of cystinuria in a cohort of 12 patients
dc.contributor.none.fl_str_mv RepositóriUM - Universidade do Minho
dc.creator.none.fl_str_mv Barbosa, Mafalda Fernanda Cabral Santos
Lopes, A.
Mota, C.
Martins, E.
Oliveira, J.
Alves, S.
Bonis, P. de
Mota, M. do Céu
Dias, C.
Santos, P. Rodrigues
Fortuna, A. M.
Quelhas, D.
Lacerda, L.
Biscegliah, L.
Cardoso, M. L.
dc.date.Accepted.fl_str_mv 2012-01-01T00:00:00Z
dc.date.available.fl_str_mv 2015-02-05T12:07:21Z
dc.date.embargoed.fl_str_mv 2015-02-05T12:07:21Z
dc.format.none.fl_str_mv application/pdf
dc.identifier.none.fl_str_mv https://hdl.handle.net/1822/33600
dc.language.none.fl_str_mv eng
dc.publisher.none.fl_str_mv John Wiley and Sons
dc.rights.none.fl_str_mv http://purl.org/coar/access_right/c_16ec
dc.subject.none.fl_str_mv Cystinuria
MLPA analysis
silent mutation
SLC3A1 gene
SLC7A9 gene
dc.title.fl_str_mv Clinical, biochemical and molecular characterization of cystinuria in a cohort of 12 patients
dc.type.none.fl_str_mv http://purl.org/coar/resource_type/c_6501
description Cystinuria is a rare autosomal inherited disorder characterized by impaired transport of cystine and dibasic aminoacids in the proximal renal tubule. Classically, cystinuria is classified as type I (silent heterozygotes) and non-type I (heterozygotes with urinary hyperexcretion of cystine). Molecularly, cystinuria is classified as type A (mutations on SLC3A1 gene) and type B (mutations on SLC7A9 gene). The goal of this study is to provide a comprehensive clinical, biochemical and molecular characterization of a cohort of 12 Portuguese patients affected with cystinuria in order to provide insight into genotype-phenotype correlations. We describe seven type I and five non-type I patients. Regarding the molecular classification, seven patients were type A and five were type B. In SLC3A1 gene, two large genomic rearrangements and 13 sequence variants, including four new variants c.611-2A>C; c.1136+44G>A; c.1597T (p.Y533N); c.*70A>G, were found. One large genomic rearrangement was found in SLC7A9 gene as well as 24 sequence variants including 3 novel variants: c.216C>T (p.C72C), c.1119G>A (p.S373S) and c.*82C>T. In our cohort the most frequent pathogenic mutations were: large rearrangements (33.3% of mutant alleles) and a missense mutation c.1400T>C (p.M467T) (11.1%). This report expands the spectrum of SLC3A1 and SLC7A9 mutations and provides guidance in the clinical implementation of molecular assays in routine genetic counseling of Portuguese patients affected with cystinuria.
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eu_rights_str_mv restrictedAccess
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fulltext.url.fl_str_mv https://repositorium.uminho.pt/bitstreams/ac1986d4-8218-471d-b71b-31a097f7c388/download
id rum_480620a163bea4abd4d20e8a2fd4e070
identifier.url.fl_str_mv https://hdl.handle.net/1822/33600
instacron_str repositorium
institution Universidade do Minho
instname_str Universidade do Minho
language eng
network_acronym_str rum
network_name_str RepositóriUM - Universidade do Minho
oai_identifier_str oai:repositorium.uminho.pt:1822/33600
organization_str_mv urn:organizationAcronym:repositorium
person_str_mv Barbosa, Mafalda Fernanda Cabral Santos
Lopes, A.
Mota, C.
Martins, E.
Oliveira, J.
Alves, S.
Bonis, P. de
Mota, M. do Céu
Dias, C.
Santos, P. Rodrigues
Fortuna, A. M.
Quelhas, D.
Lacerda, L.
Biscegliah, L.
Cardoso, M. L.
publishDate 2012
publisher.none.fl_str_mv John Wiley and Sons
reponame_str RepositóriUM - Universidade do Minho
repository_id_str urn:repositoryAcronym:rum
service_str_mv urn:repositoryAcronym:rum
spelling engJohn Wiley and SonsporCystinuria is a rare autosomal inherited disorder characterized by impaired transport of cystine and dibasic aminoacids in the proximal renal tubule. Classically, cystinuria is classified as type I (silent heterozygotes) and non-type I (heterozygotes with urinary hyperexcretion of cystine). Molecularly, cystinuria is classified as type A (mutations on SLC3A1 gene) and type B (mutations on SLC7A9 gene). The goal of this study is to provide a comprehensive clinical, biochemical and molecular characterization of a cohort of 12 Portuguese patients affected with cystinuria in order to provide insight into genotype-phenotype correlations. We describe seven type I and five non-type I patients. Regarding the molecular classification, seven patients were type A and five were type B. In SLC3A1 gene, two large genomic rearrangements and 13 sequence variants, including four new variants c.611-2A>C; c.1136+44G>A; c.1597T (p.Y533N); c.*70A>G, were found. One large genomic rearrangement was found in SLC7A9 gene as well as 24 sequence variants including 3 novel variants: c.216C>T (p.C72C), c.1119G>A (p.S373S) and c.*82C>T. In our cohort the most frequent pathogenic mutations were: large rearrangements (33.3% of mutant alleles) and a missense mutation c.1400T>C (p.M467T) (11.1%). This report expands the spectrum of SLC3A1 and SLC7A9 mutations and provides guidance in the clinical implementation of molecular assays in routine genetic counseling of Portuguese patients affected with cystinuria.application/pdfporClinical, biochemical and molecular characterization of cystinuria in a cohort of 12 patientsBarbosa, Mafalda Fernanda Cabral SantosLopes, A.Mota, C.Martins, E.Oliveira, J.Alves, S.Bonis, P. deMota, M. do CéuDias, C.Santos, P. RodriguesFortuna, A. M.Quelhas, D.Lacerda, L.Biscegliah, L.Cardoso, M. L.HostingInstitutionOrganizationalRepositóriUM - Universidade do Minhoe-mailmailto:repositorium@usdb.uminho.ptrepositorium@usdb.uminho.ptPMID21255007ISSNIsPartOf0009-9163DOIIsPartOf10.1111/j.1399-0004.2011.01638.x2015-02-05T12:07:21Z20122015-01-30T16:25:53Z2012-01-01T00:00:00ZHandlehttps://hdl.handle.net/1822/33600http://purl.org/coar/access_right/c_16ecrestricted accessCystinuriaMLPA analysissilent mutationSLC3A1 geneSLC7A9 gene581016 bytesliteraturehttp://purl.org/coar/resource_type/c_6501journal articlehttp://purl.org/coar/access_right/c_16ecapplication/pdffulltexthttps://repositorium.uminho.pt/bitstreams/ac1986d4-8218-471d-b71b-31a097f7c388/download
spellingShingle Clinical, biochemical and molecular characterization of cystinuria in a cohort of 12 patients
Barbosa, Mafalda Fernanda Cabral Santos
Cystinuria
MLPA analysis
silent mutation
SLC3A1 gene
SLC7A9 gene
status SINGLETON
subject.fl_str_mv Cystinuria
MLPA analysis
silent mutation
SLC3A1 gene
SLC7A9 gene
title Clinical, biochemical and molecular characterization of cystinuria in a cohort of 12 patients
title_full Clinical, biochemical and molecular characterization of cystinuria in a cohort of 12 patients
title_fullStr Clinical, biochemical and molecular characterization of cystinuria in a cohort of 12 patients
title_full_unstemmed Clinical, biochemical and molecular characterization of cystinuria in a cohort of 12 patients
title_short Clinical, biochemical and molecular characterization of cystinuria in a cohort of 12 patients
title_sort Clinical, biochemical and molecular characterization of cystinuria in a cohort of 12 patients
topic Cystinuria
MLPA analysis
silent mutation
SLC3A1 gene
SLC7A9 gene
topic_facet Cystinuria
MLPA analysis
silent mutation
SLC3A1 gene
SLC7A9 gene
url https://hdl.handle.net/1822/33600
visible 1