Publicação

CD4+ T cell vaccination overcomes defective cross-presentation of fungal antigens in a mouse model of chronic granulomatous disease

Ver documento

Detalhes bibliográficos
Resumo:Aspergillus fumigatus is a model fungal pathogen and a common cause of infection in individuals with the primary immunodeficiency chronic granulomatous disease (CGD). Although primarily considered a deficiency of innate immunity, CGD is also linked to dysfunctional T cell reactivity. Both CD4+ and CD8+ T cells mediate vaccine-induced protection from experimental aspergillosis, but the molecular mechanisms leading to the generation of protective immunity and whether these mechanisms are dysregulated in individuals with CGD have not been determined. Here, we show that activation of either T cell subset in a mouse model of CGD is contingent upon the nature of the fungal vaccine, the involvement of distinct innate receptor signaling pathways, and the mode of antigen routing and presentation in DCs. Aspergillus conidia activated CD8+ T cells upon sorting to the Rab14+ endosomal compartment required for alternative MHC class I presentation. Cross-priming of CD8+ T cells failed to occur in mice with CGD due to defective DC endosomal alkalinization and autophagy. However, long-lasting antifungal protection and disease control were successfully achieved upon vaccination with purified fungal antigens that activated CD4+ T cells through the endosome/lysosome pathway. Our study thus indicates that distinct intracellular pathways are exploited for the priming of CD4+ and CD8+ T cells to A. fumigatus and suggests that CD4+ T cell vaccination may be able to overcome defective antifungal CD8+ T cell memory in individuals with CGD.
Autores principais:Luca, Antonella De
Outros Autores:Iannitti, Rossana G.; Bozza, Silvia; Beau, Remi; Casagrande, Andrea; D’Angelo, Carmen; Moretti, Silvia; Cunha, Cristina; Giovannini, Gloria; Massi-Benedetti, Cristina; Carvalho, Agostinho; Boon, Louis; Latgé, Jean-Paul; Romani, Luigina
Ano:2012
País:Portugal
Tipo de documento:artigo
Tipo de acesso:acesso aberto
Instituição associada:Universidade do Minho
Idioma:inglês
Origem:RepositóriUM - Universidade do Minho
_version_ 1867439309621035008
author Luca, Antonella De
author2 Iannitti, Rossana G.
Bozza, Silvia
Beau, Remi
Casagrande, Andrea
D’Angelo, Carmen
Moretti, Silvia
Cunha, Cristina
Giovannini, Gloria
Massi-Benedetti, Cristina
Carvalho, Agostinho
Boon, Louis
Latgé, Jean-Paul
Romani, Luigina
author2_role author
author
author
author
author
author
author
author
author
author
author
author
author
author_facet Luca, Antonella De
Iannitti, Rossana G.
Bozza, Silvia
Beau, Remi
Casagrande, Andrea
D’Angelo, Carmen
Moretti, Silvia
Cunha, Cristina
Giovannini, Gloria
Massi-Benedetti, Cristina
Carvalho, Agostinho
Boon, Louis
Latgé, Jean-Paul
Romani, Luigina
author_role author
contributor_name_str_mv RepositóriUM - Universidade do Minho
country_str PT
creators_json_txt [{\"Person.name\":\"Luca, Antonella De\"},{\"Person.name\":\"Iannitti, Rossana G.\"},{\"Person.name\":\"Bozza, Silvia\"},{\"Person.name\":\"Beau, Remi\"},{\"Person.name\":\"Casagrande, Andrea\"},{\"Person.name\":\"D’Angelo, Carmen\"},{\"Person.name\":\"Moretti, Silvia\"},{\"Person.name\":\"Cunha, Cristina\"},{\"Person.name\":\"Giovannini, Gloria\"},{\"Person.name\":\"Massi-Benedetti, Cristina\"},{\"Person.name\":\"Carvalho, Agostinho\"},{\"Person.name\":\"Boon, Louis\"},{\"Person.name\":\"Latgé, Jean-Paul\"},{\"Person.name\":\"Romani, Luigina\"}]
datacite.contributors.contributor.contributorName.fl_str_mv RepositóriUM - Universidade do Minho
datacite.creators.creator.creatorName.fl_str_mv Luca, Antonella De
Iannitti, Rossana G.
Bozza, Silvia
Beau, Remi
Casagrande, Andrea
D’Angelo, Carmen
Moretti, Silvia
Cunha, Cristina
Giovannini, Gloria
Massi-Benedetti, Cristina
Carvalho, Agostinho
Boon, Louis
Latgé, Jean-Paul
Romani, Luigina
datacite.date.Accepted.fl_str_mv 2012-05-01T00:00:00Z
datacite.date.available.fl_str_mv 2013-05-03T11:21:46Z
datacite.date.embargoed.fl_str_mv 2013-05-03T11:21:46Z
datacite.rights.fl_str_mv http://purl.org/coar/access_right/c_abf2
datacite.titles.title.fl_str_mv CD4+ T cell vaccination overcomes defective cross-presentation of fungal antigens in a mouse model of chronic granulomatous disease
dc.contributor.none.fl_str_mv RepositóriUM - Universidade do Minho
dc.creator.none.fl_str_mv Luca, Antonella De
Iannitti, Rossana G.
Bozza, Silvia
Beau, Remi
Casagrande, Andrea
D’Angelo, Carmen
Moretti, Silvia
Cunha, Cristina
Giovannini, Gloria
Massi-Benedetti, Cristina
Carvalho, Agostinho
Boon, Louis
Latgé, Jean-Paul
Romani, Luigina
dc.date.Accepted.fl_str_mv 2012-05-01T00:00:00Z
dc.date.available.fl_str_mv 2013-05-03T11:21:46Z
dc.date.embargoed.fl_str_mv 2013-05-03T11:21:46Z
dc.format.none.fl_str_mv application/pdf
dc.identifier.none.fl_str_mv https://hdl.handle.net/1822/23919
dc.language.none.fl_str_mv eng
dc.publisher.none.fl_str_mv American Society for Clinical Investigation
dc.rights.none.fl_str_mv http://purl.org/coar/access_right/c_abf2
dc.title.fl_str_mv CD4+ T cell vaccination overcomes defective cross-presentation of fungal antigens in a mouse model of chronic granulomatous disease
dc.type.none.fl_str_mv http://purl.org/coar/resource_type/c_6501
description Aspergillus fumigatus is a model fungal pathogen and a common cause of infection in individuals with the primary immunodeficiency chronic granulomatous disease (CGD). Although primarily considered a deficiency of innate immunity, CGD is also linked to dysfunctional T cell reactivity. Both CD4+ and CD8+ T cells mediate vaccine-induced protection from experimental aspergillosis, but the molecular mechanisms leading to the generation of protective immunity and whether these mechanisms are dysregulated in individuals with CGD have not been determined. Here, we show that activation of either T cell subset in a mouse model of CGD is contingent upon the nature of the fungal vaccine, the involvement of distinct innate receptor signaling pathways, and the mode of antigen routing and presentation in DCs. Aspergillus conidia activated CD8+ T cells upon sorting to the Rab14+ endosomal compartment required for alternative MHC class I presentation. Cross-priming of CD8+ T cells failed to occur in mice with CGD due to defective DC endosomal alkalinization and autophagy. However, long-lasting antifungal protection and disease control were successfully achieved upon vaccination with purified fungal antigens that activated CD4+ T cells through the endosome/lysosome pathway. Our study thus indicates that distinct intracellular pathways are exploited for the priming of CD4+ and CD8+ T cells to A. fumigatus and suggests that CD4+ T cell vaccination may be able to overcome defective antifungal CD8+ T cell memory in individuals with CGD.
dirty 0
eu_rights_str_mv openAccess
format article
fulltext.url.fl_str_mv https://repositorium.uminho.pt/bitstreams/1bffc6da-6bf1-4fe6-9844-30d5cab96f32/download
id rum_4c8bcb984e2c74ea64b331b6bc538227
identifier.url.fl_str_mv https://hdl.handle.net/1822/23919
instacron_str repositorium
institution Universidade do Minho
instname_str Universidade do Minho
language eng
network_acronym_str rum
network_name_str RepositóriUM - Universidade do Minho
oai_identifier_str oai:repositorium.uminho.pt:1822/23919
organization_str_mv urn:organizationAcronym:repositorium
person_str_mv Luca, Antonella De
Iannitti, Rossana G.
Bozza, Silvia
Beau, Remi
Casagrande, Andrea
D’Angelo, Carmen
Moretti, Silvia
Cunha, Cristina
Giovannini, Gloria
Massi-Benedetti, Cristina
Carvalho, Agostinho
Boon, Louis
Latgé, Jean-Paul
Romani, Luigina
publishDate 2012
publisher.none.fl_str_mv American Society for Clinical Investigation
reponame_str RepositóriUM - Universidade do Minho
repository_id_str urn:repositoryAcronym:rum
service_str_mv urn:repositoryAcronym:rum
spelling engAmerican Society for Clinical InvestigationporAspergillus fumigatus is a model fungal pathogen and a common cause of infection in individuals with the primary immunodeficiency chronic granulomatous disease (CGD). Although primarily considered a deficiency of innate immunity, CGD is also linked to dysfunctional T cell reactivity. Both CD4+ and CD8+ T cells mediate vaccine-induced protection from experimental aspergillosis, but the molecular mechanisms leading to the generation of protective immunity and whether these mechanisms are dysregulated in individuals with CGD have not been determined. Here, we show that activation of either T cell subset in a mouse model of CGD is contingent upon the nature of the fungal vaccine, the involvement of distinct innate receptor signaling pathways, and the mode of antigen routing and presentation in DCs. Aspergillus conidia activated CD8+ T cells upon sorting to the Rab14+ endosomal compartment required for alternative MHC class I presentation. Cross-priming of CD8+ T cells failed to occur in mice with CGD due to defective DC endosomal alkalinization and autophagy. However, long-lasting antifungal protection and disease control were successfully achieved upon vaccination with purified fungal antigens that activated CD4+ T cells through the endosome/lysosome pathway. Our study thus indicates that distinct intracellular pathways are exploited for the priming of CD4+ and CD8+ T cells to A. fumigatus and suggests that CD4+ T cell vaccination may be able to overcome defective antifungal CD8+ T cell memory in individuals with CGD.application/pdfporCD4+ T cell vaccination overcomes defective cross-presentation of fungal antigens in a mouse model of chronic granulomatous diseaseLuca, Antonella DeIannitti, Rossana G.Bozza, SilviaBeau, RemiCasagrande, AndreaD’Angelo, CarmenMoretti, SilviaCunha, CristinaGiovannini, GloriaMassi-Benedetti, CristinaCarvalho, AgostinhoBoon, LouisLatgé, Jean-PaulRomani, LuiginaHostingInstitutionOrganizationalRepositóriUM - Universidade do Minhoe-mailmailto:repositorium@usdb.uminho.ptrepositorium@usdb.uminho.ptPMID22523066ISSNIsPartOf0021-9738DOIIsPartOf10.1172/JCI608622013-05-03T11:21:46Z2012-052012-05-01T00:00:00ZHandlehttps://hdl.handle.net/1822/23919http://purl.org/coar/access_right/c_abf2open access5940842 bytesliteraturehttp://purl.org/coar/resource_type/c_6501journal articlehttp://purl.org/coar/access_right/c_abf2application/pdffulltexthttps://repositorium.uminho.pt/bitstreams/1bffc6da-6bf1-4fe6-9844-30d5cab96f32/download
spellingShingle CD4+ T cell vaccination overcomes defective cross-presentation of fungal antigens in a mouse model of chronic granulomatous disease
Luca, Antonella De
status SINGLETON
title CD4+ T cell vaccination overcomes defective cross-presentation of fungal antigens in a mouse model of chronic granulomatous disease
title_full CD4+ T cell vaccination overcomes defective cross-presentation of fungal antigens in a mouse model of chronic granulomatous disease
title_fullStr CD4+ T cell vaccination overcomes defective cross-presentation of fungal antigens in a mouse model of chronic granulomatous disease
title_full_unstemmed CD4+ T cell vaccination overcomes defective cross-presentation of fungal antigens in a mouse model of chronic granulomatous disease
title_short CD4+ T cell vaccination overcomes defective cross-presentation of fungal antigens in a mouse model of chronic granulomatous disease
title_sort CD4+ T cell vaccination overcomes defective cross-presentation of fungal antigens in a mouse model of chronic granulomatous disease
url https://hdl.handle.net/1822/23919
visible 1