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Exploring the anticancer effect of new ruthenium-biotinylated conjugates in colorectal cancer

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Resumo:Colorectal cancer (CRC) is one of the most commonly diagnosed cancers and a leading cause of cancer death worldwide. The current chemotherapeutic agents available for CRC treatment are limited, accompanied by severe side effects and resistance acquisition. Ruthenium (Ru) compounds have been explored because they encompass several anticancer properties that make them promising therapeutic agents. Considering this, two new ruthenium-biotinylated conjugates (LCR134 and LCR220) were recently synthesized, in an attempt to bring more efficiency and selectivity to target cancer cells. LCR134 comprises a Ru cytotoxic fragment, with the addition of two molecules of biotin to the organometallic center (active targeting), while LCR220 has a polymeric chain, with two biotin molecules at the end (passive and active targeting). The aim of this work was to determine whether these Ru-biotinylated conjugates are promising candidates for CRC therapy. In this work, the effects of the newly Ru agents were assessed in CRC cells (SW480 and RKO) and in a “normal” colon-derived cell line (NCM460). In order to achieve our aim, we studied the effect of Ru compounds on cell viability, cell death, F-actin cytoskeleton structure, as well as in β-actin and glucose transporters 1 (GLUT1) expression levels. The results showed that the compounds were more cytotoxic for CRC cell lines in comparison with the normal colon cells. Moreover, all the half-maximum inhibitory concentration (IC50) values with the exception of LCR134 for SW480 cells, were lower than the values of the classical chemotherapeutic agent cisplatin, suggesting a higher efficacy of our ruthenium-biotinylated conjugates towards CRC cells. Ru compounds also interfere with the clonogenic ability of both CRC cell lines, without inducing cell death in a significant manner, suggesting that their mechanism of action affects proliferation. Additionally, these agents induced structural changes in the cytoskeleton, namely in F-actin filaments. We could not observe alterations on the expression levels of β-actin and GLUT1. Our results highlight the promising anticancer activities of these agents towards CRC cells.
Autores principais:Fernandes, Pedro José Pereira
Assunto:Actin cytoskeleton Clonogenic ability Colorectal cancer Ruthenium Cancro colorretal Capacidade clonogénica Citoesqueleto de actina Ruténio
Ano:2020
País:Portugal
Tipo de documento:dissertação de mestrado
Tipo de acesso:acesso aberto
Instituição associada:Universidade do Minho
Idioma:inglês
Origem:RepositóriUM - Universidade do Minho
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author Fernandes, Pedro José Pereira
author_facet Fernandes, Pedro José Pereira
author_role author
contributor_name_str_mv Preto, Ana
Universidade do Minho
country_str PT
creators_json_txt [{\"Person.name\":\"Fernandes, Pedro José Pereira\"}]
datacite.contributors.contributor.contributorName.fl_str_mv Preto, Ana
Universidade do Minho
datacite.creators.creator.creatorName.fl_str_mv Fernandes, Pedro José Pereira
datacite.date.Accepted.fl_str_mv 2020-01-28T00:00:00Z
datacite.date.available.fl_str_mv 2022-11-08T12:00:07Z
datacite.date.embargoed.fl_str_mv 2022-11-08T12:00:07Z
datacite.rights.fl_str_mv http://purl.org/coar/access_right/c_abf2
datacite.subjects.subject.fl_str_mv Actin cytoskeleton
Clonogenic ability
Colorectal cancer
Ruthenium
Cancro colorretal
Capacidade clonogénica
Citoesqueleto de actina
Ruténio
datacite.titles.title.fl_str_mv Exploring the anticancer effect of new ruthenium-biotinylated conjugates in colorectal cancer
Explorar o efeito anticancerígeno de novos conjugados de ruténio-biotinilados no cancro colorretal
dc.contributor.none.fl_str_mv Preto, Ana
Universidade do Minho
dc.creator.none.fl_str_mv Fernandes, Pedro José Pereira
dc.date.Accepted.fl_str_mv 2020-01-28T00:00:00Z
dc.date.available.fl_str_mv 2022-11-08T12:00:07Z
dc.date.embargoed.fl_str_mv 2022-11-08T12:00:07Z
dc.format.none.fl_str_mv application/pdf
dc.identifier.none.fl_str_mv https://hdl.handle.net/1822/80492
dc.language.none.fl_str_mv eng
dc.rights.cclincense.fl_str_mv http://creativecommons.org/licenses/by-nc-nd/4.0/
dc.rights.none.fl_str_mv http://purl.org/coar/access_right/c_abf2
dc.rights.rights.copyright.fl_str_mv openAccess
dc.subject.none.fl_str_mv Actin cytoskeleton
Clonogenic ability
Colorectal cancer
Ruthenium
Cancro colorretal
Capacidade clonogénica
Citoesqueleto de actina
Ruténio
dc.title.fl_str_mv Exploring the anticancer effect of new ruthenium-biotinylated conjugates in colorectal cancer
Explorar o efeito anticancerígeno de novos conjugados de ruténio-biotinilados no cancro colorretal
dc.type.none.fl_str_mv http://purl.org/coar/resource_type/c_bdcc
description Colorectal cancer (CRC) is one of the most commonly diagnosed cancers and a leading cause of cancer death worldwide. The current chemotherapeutic agents available for CRC treatment are limited, accompanied by severe side effects and resistance acquisition. Ruthenium (Ru) compounds have been explored because they encompass several anticancer properties that make them promising therapeutic agents. Considering this, two new ruthenium-biotinylated conjugates (LCR134 and LCR220) were recently synthesized, in an attempt to bring more efficiency and selectivity to target cancer cells. LCR134 comprises a Ru cytotoxic fragment, with the addition of two molecules of biotin to the organometallic center (active targeting), while LCR220 has a polymeric chain, with two biotin molecules at the end (passive and active targeting). The aim of this work was to determine whether these Ru-biotinylated conjugates are promising candidates for CRC therapy. In this work, the effects of the newly Ru agents were assessed in CRC cells (SW480 and RKO) and in a “normal” colon-derived cell line (NCM460). In order to achieve our aim, we studied the effect of Ru compounds on cell viability, cell death, F-actin cytoskeleton structure, as well as in β-actin and glucose transporters 1 (GLUT1) expression levels. The results showed that the compounds were more cytotoxic for CRC cell lines in comparison with the normal colon cells. Moreover, all the half-maximum inhibitory concentration (IC50) values with the exception of LCR134 for SW480 cells, were lower than the values of the classical chemotherapeutic agent cisplatin, suggesting a higher efficacy of our ruthenium-biotinylated conjugates towards CRC cells. Ru compounds also interfere with the clonogenic ability of both CRC cell lines, without inducing cell death in a significant manner, suggesting that their mechanism of action affects proliferation. Additionally, these agents induced structural changes in the cytoskeleton, namely in F-actin filaments. We could not observe alterations on the expression levels of β-actin and GLUT1. Our results highlight the promising anticancer activities of these agents towards CRC cells.
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format masterThesis
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id rum_eb5918dd7cd5ce8ea505d237b59ec30d
identifier.url.fl_str_mv https://hdl.handle.net/1822/80492
instacron_str repositorium
institution Universidade do Minho
instname_str Universidade do Minho
language eng
network_acronym_str rum
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oai_identifier_str oai:repositorium.uminho.pt:1822/80492
organization_str_mv urn:organizationAcronym:repositorium
person_str_mv Fernandes, Pedro José Pereira
publishDate 2020
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spelling engporColorectal cancer (CRC) is one of the most commonly diagnosed cancers and a leading cause of cancer death worldwide. The current chemotherapeutic agents available for CRC treatment are limited, accompanied by severe side effects and resistance acquisition. Ruthenium (Ru) compounds have been explored because they encompass several anticancer properties that make them promising therapeutic agents. Considering this, two new ruthenium-biotinylated conjugates (LCR134 and LCR220) were recently synthesized, in an attempt to bring more efficiency and selectivity to target cancer cells. LCR134 comprises a Ru cytotoxic fragment, with the addition of two molecules of biotin to the organometallic center (active targeting), while LCR220 has a polymeric chain, with two biotin molecules at the end (passive and active targeting). The aim of this work was to determine whether these Ru-biotinylated conjugates are promising candidates for CRC therapy. In this work, the effects of the newly Ru agents were assessed in CRC cells (SW480 and RKO) and in a “normal” colon-derived cell line (NCM460). In order to achieve our aim, we studied the effect of Ru compounds on cell viability, cell death, F-actin cytoskeleton structure, as well as in β-actin and glucose transporters 1 (GLUT1) expression levels. The results showed that the compounds were more cytotoxic for CRC cell lines in comparison with the normal colon cells. Moreover, all the half-maximum inhibitory concentration (IC50) values with the exception of LCR134 for SW480 cells, were lower than the values of the classical chemotherapeutic agent cisplatin, suggesting a higher efficacy of our ruthenium-biotinylated conjugates towards CRC cells. Ru compounds also interfere with the clonogenic ability of both CRC cell lines, without inducing cell death in a significant manner, suggesting that their mechanism of action affects proliferation. Additionally, these agents induced structural changes in the cytoskeleton, namely in F-actin filaments. We could not observe alterations on the expression levels of β-actin and GLUT1. Our results highlight the promising anticancer activities of these agents towards CRC cells.application/pdfporExploring the anticancer effect of new ruthenium-biotinylated conjugates in colorectal cancerAlternativeTitleporExplorar o efeito anticancerígeno de novos conjugados de ruténio-biotinilados no cancro colorretalFernandes, Pedro José PereiraPreto, AnaHostingInstitutionOrganizationalUniversidade do Minhoe-mailmailto:repositorium@usdb.uminho.ptrepositorium@usdb.uminho.ptURNurn:tid:2024741352022-11-08T12:00:07Z2020-01-282019-102020-01-28T00:00:00ZHandlehttps://hdl.handle.net/1822/80492http://purl.org/coar/access_right/c_abf2open accessActin cytoskeletonClonogenic abilityColorectal cancerRutheniumCancro colorretalCapacidade clonogénicaCitoesqueleto de actinaRuténio4114620 bytesliteraturehttp://purl.org/coar/resource_type/c_bdccmaster thesis2020-01-28http://creativecommons.org/licenses/by-nc-nd/4.0/openAccesshttp://purl.org/coar/access_right/c_abf2application/pdffulltexthttps://prod-dspace.uminho.pt/bitstreams/ec8a547d-f6ca-4854-9144-7950016029fa/download
spellingShingle Exploring the anticancer effect of new ruthenium-biotinylated conjugates in colorectal cancer
Fernandes, Pedro José Pereira
Actin cytoskeleton
Clonogenic ability
Colorectal cancer
Ruthenium
Cancro colorretal
Capacidade clonogénica
Citoesqueleto de actina
Ruténio
status SINGLETON
subject.fl_str_mv Actin cytoskeleton
Clonogenic ability
Colorectal cancer
Ruthenium
Cancro colorretal
Capacidade clonogénica
Citoesqueleto de actina
Ruténio
title Exploring the anticancer effect of new ruthenium-biotinylated conjugates in colorectal cancer
title_full Exploring the anticancer effect of new ruthenium-biotinylated conjugates in colorectal cancer
title_fullStr Exploring the anticancer effect of new ruthenium-biotinylated conjugates in colorectal cancer
title_full_unstemmed Exploring the anticancer effect of new ruthenium-biotinylated conjugates in colorectal cancer
title_short Exploring the anticancer effect of new ruthenium-biotinylated conjugates in colorectal cancer
title_sort Exploring the anticancer effect of new ruthenium-biotinylated conjugates in colorectal cancer
topic Actin cytoskeleton
Clonogenic ability
Colorectal cancer
Ruthenium
Cancro colorretal
Capacidade clonogénica
Citoesqueleto de actina
Ruténio
topic_facet Actin cytoskeleton
Clonogenic ability
Colorectal cancer
Ruthenium
Cancro colorretal
Capacidade clonogénica
Citoesqueleto de actina
Ruténio
url https://hdl.handle.net/1822/80492
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