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Evidence for an intramacrophage growth phase of Mycobacterium ulcerans

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Resumo:Mycobacterium ulcerans is the etiologic agent of Buruli ulcer (BU), an emerging tropical skin disease. Virulent M. ulcerans secretes mycolactone, a cytotoxic exotoxin with a key pathogenic role. M. ulcerans in biopsy specimens has been described as an extracellular bacillus. In vitro assays have suggested a mycolactone-induced inhibition of M. ulcerans uptake by macrophages in which its proliferation has not been demonstrated. Therefore, and uniquely for a mycobacterium, M. ulcerans has been classified as an extracellular pathogen. In specimens from patients and in mouse footpad lesions, extracellular bacilli were concentrated in central necrotic acellular areas; however, we found bacilli within macrophages in surrounding inflammatory infiltrates. We demonstrated that mycolactone-producing M. ulcerans isolates are efficiently phagocytosed by murine macrophages, indicating that the extracellular location of M. ulcerans is not a result of inhibition of phagocytosis. Additionally, we found that M. ulcerans multiplies inside cultured mouse macrophages when low multiplicities of infection are used to prevent early mycolactone-associated cytotoxicity. Following the proliferation phase within macrophages, M. ulcerans induces the lysis of the infected host cells, becoming extracellular. Our data show that M. ulcerans, like M. tuberculosis, is an intracellular parasite with phases of intramacrophage and extracellular multiplication. The occurrence of an intramacrophage phase is in accordance with the development of cell-mediated and delayed-type hypersensitivity responses in BU patients.
Autores principais:Torrado, Egídio
Outros Autores:Fraga, Alexandra Gabriel; Castro, António G.; Stragier, Pieter; Meyers, Wayne M.; Portaels, Françoise; Silva, Manuel T.; Pedrosa, Jorge
Assunto:Animals Bacterial Toxins Cells, Cultured Disease Models, Animal Female Foot Histocytochemistry Humans Macrolides Macrophages Mice Mice, Inbred BALB C Microscopy, Electron, Transmission Mycobacterium Infections, Nontuberculous Mycobacterium ulcerans Phagocytosis Skin Diseases, Bacterial Skin Ulcer
Ano:2007
País:Portugal
Tipo de documento:artigo
Tipo de acesso:acesso aberto
Instituição associada:Universidade do Minho
Idioma:inglês
Origem:RepositóriUM - Universidade do Minho
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author Torrado, Egídio
author2 Fraga, Alexandra Gabriel
Castro, António G.
Stragier, Pieter
Meyers, Wayne M.
Portaels, Françoise
Silva, Manuel T.
Pedrosa, Jorge
author2_role author
author
author
author
author
author
author
author_facet Torrado, Egídio
Fraga, Alexandra Gabriel
Castro, António G.
Stragier, Pieter
Meyers, Wayne M.
Portaels, Françoise
Silva, Manuel T.
Pedrosa, Jorge
author_role author
contributor_name_str_mv Universidade do Minho
country_str PT
creators_json_txt [{\"Person.name\":\"Torrado, Egídio\"},{\"Person.name\":\"Fraga, Alexandra Gabriel\"},{\"Person.name\":\"Castro, António G.\"},{\"Person.name\":\"Stragier, Pieter\"},{\"Person.name\":\"Meyers, Wayne M.\"},{\"Person.name\":\"Portaels, Françoise\"},{\"Person.name\":\"Silva, Manuel T.\"},{\"Person.name\":\"Pedrosa, Jorge\"}]
datacite.contributors.contributor.contributorName.fl_str_mv Universidade do Minho
datacite.creators.creator.creatorName.fl_str_mv Torrado, Egídio
Fraga, Alexandra Gabriel
Castro, António G.
Stragier, Pieter
Meyers, Wayne M.
Portaels, Françoise
Silva, Manuel T.
Pedrosa, Jorge
datacite.date.Accepted.fl_str_mv 2007-02-01T00:00:00Z
datacite.date.available.fl_str_mv 2020-10-27T13:25:46Z
datacite.date.embargoed.fl_str_mv 2020-10-27T13:25:46Z
datacite.rights.fl_str_mv http://purl.org/coar/access_right/c_abf2
datacite.subjects.subject.fl_str_mv Animals
Bacterial Toxins
Cells, Cultured
Disease Models, Animal
Female
Foot
Histocytochemistry
Humans
Macrolides
Macrophages
Mice
Mice, Inbred BALB C
Microscopy, Electron, Transmission
Mycobacterium Infections, Nontuberculous
Mycobacterium ulcerans
Phagocytosis
Skin Diseases, Bacterial
Skin Ulcer
datacite.titles.title.fl_str_mv Evidence for an intramacrophage growth phase of Mycobacterium ulcerans
dc.contributor.none.fl_str_mv Universidade do Minho
dc.creator.none.fl_str_mv Torrado, Egídio
Fraga, Alexandra Gabriel
Castro, António G.
Stragier, Pieter
Meyers, Wayne M.
Portaels, Françoise
Silva, Manuel T.
Pedrosa, Jorge
dc.date.Accepted.fl_str_mv 2007-02-01T00:00:00Z
dc.date.available.fl_str_mv 2020-10-27T13:25:46Z
dc.date.embargoed.fl_str_mv 2020-10-27T13:25:46Z
dc.format.none.fl_str_mv application/pdf
dc.identifier.none.fl_str_mv https://hdl.handle.net/1822/67769
dc.language.none.fl_str_mv eng
dc.publisher.none.fl_str_mv American Society for Microbiology (ASM)
dc.rights.none.fl_str_mv http://purl.org/coar/access_right/c_abf2
dc.subject.none.fl_str_mv Animals
Bacterial Toxins
Cells, Cultured
Disease Models, Animal
Female
Foot
Histocytochemistry
Humans
Macrolides
Macrophages
Mice
Mice, Inbred BALB C
Microscopy, Electron, Transmission
Mycobacterium Infections, Nontuberculous
Mycobacterium ulcerans
Phagocytosis
Skin Diseases, Bacterial
Skin Ulcer
dc.title.fl_str_mv Evidence for an intramacrophage growth phase of Mycobacterium ulcerans
dc.type.none.fl_str_mv http://purl.org/coar/resource_type/c_6501
description Mycobacterium ulcerans is the etiologic agent of Buruli ulcer (BU), an emerging tropical skin disease. Virulent M. ulcerans secretes mycolactone, a cytotoxic exotoxin with a key pathogenic role. M. ulcerans in biopsy specimens has been described as an extracellular bacillus. In vitro assays have suggested a mycolactone-induced inhibition of M. ulcerans uptake by macrophages in which its proliferation has not been demonstrated. Therefore, and uniquely for a mycobacterium, M. ulcerans has been classified as an extracellular pathogen. In specimens from patients and in mouse footpad lesions, extracellular bacilli were concentrated in central necrotic acellular areas; however, we found bacilli within macrophages in surrounding inflammatory infiltrates. We demonstrated that mycolactone-producing M. ulcerans isolates are efficiently phagocytosed by murine macrophages, indicating that the extracellular location of M. ulcerans is not a result of inhibition of phagocytosis. Additionally, we found that M. ulcerans multiplies inside cultured mouse macrophages when low multiplicities of infection are used to prevent early mycolactone-associated cytotoxicity. Following the proliferation phase within macrophages, M. ulcerans induces the lysis of the infected host cells, becoming extracellular. Our data show that M. ulcerans, like M. tuberculosis, is an intracellular parasite with phases of intramacrophage and extracellular multiplication. The occurrence of an intramacrophage phase is in accordance with the development of cell-mediated and delayed-type hypersensitivity responses in BU patients.
dirty 0
eu_rights_str_mv openAccess
format article
fulltext.url.fl_str_mv https://prod-dspace.uminho.pt/bitstreams/a2a239e7-64ff-4186-90c3-df867fbc1361/download
id rum_ebfc084ab9f25bbb0a43c64ea686b7bc
identifier.url.fl_str_mv https://hdl.handle.net/1822/67769
instacron_str repositorium
institution Universidade do Minho
instname_str Universidade do Minho
language eng
network_acronym_str rum
network_name_str RepositóriUM - Universidade do Minho
oai_identifier_str oai:repositorium.uminho.pt:1822/67769
organization_str_mv urn:organizationAcronym:repositorium
person_str_mv Torrado, Egídio
Fraga, Alexandra Gabriel
Castro, António G.
Stragier, Pieter
Meyers, Wayne M.
Portaels, Françoise
Silva, Manuel T.
Pedrosa, Jorge
publishDate 2007
publisher.none.fl_str_mv American Society for Microbiology (ASM)
reponame_str RepositóriUM - Universidade do Minho
repository_id_str urn:repositoryAcronym:rum
service_str_mv urn:repositoryAcronym:rum
spelling engAmerican Society for Microbiology (ASM)porMycobacterium ulcerans is the etiologic agent of Buruli ulcer (BU), an emerging tropical skin disease. Virulent M. ulcerans secretes mycolactone, a cytotoxic exotoxin with a key pathogenic role. M. ulcerans in biopsy specimens has been described as an extracellular bacillus. In vitro assays have suggested a mycolactone-induced inhibition of M. ulcerans uptake by macrophages in which its proliferation has not been demonstrated. Therefore, and uniquely for a mycobacterium, M. ulcerans has been classified as an extracellular pathogen. In specimens from patients and in mouse footpad lesions, extracellular bacilli were concentrated in central necrotic acellular areas; however, we found bacilli within macrophages in surrounding inflammatory infiltrates. We demonstrated that mycolactone-producing M. ulcerans isolates are efficiently phagocytosed by murine macrophages, indicating that the extracellular location of M. ulcerans is not a result of inhibition of phagocytosis. Additionally, we found that M. ulcerans multiplies inside cultured mouse macrophages when low multiplicities of infection are used to prevent early mycolactone-associated cytotoxicity. Following the proliferation phase within macrophages, M. ulcerans induces the lysis of the infected host cells, becoming extracellular. Our data show that M. ulcerans, like M. tuberculosis, is an intracellular parasite with phases of intramacrophage and extracellular multiplication. The occurrence of an intramacrophage phase is in accordance with the development of cell-mediated and delayed-type hypersensitivity responses in BU patients.application/pdfporEvidence for an intramacrophage growth phase of Mycobacterium ulceransTorrado, EgídioFraga, Alexandra GabrielCastro, António G.Stragier, PieterMeyers, Wayne M.Portaels, FrançoiseSilva, Manuel T.Pedrosa, JorgeHostingInstitutionOrganizationalUniversidade do Minhoe-mailmailto:repositorium@usdb.uminho.ptrepositorium@usdb.uminho.ptISSNIsPartOf0019-9567DOIIsPartOf10.1128/IAI.00889-062020-10-27T13:25:46Z2007-022007-02-01T00:00:00ZHandlehttps://hdl.handle.net/1822/67769http://purl.org/coar/access_right/c_abf2open accessAnimalsBacterial ToxinsCells, CulturedDisease Models, AnimalFemaleFootHistocytochemistryHumansMacrolidesMacrophagesMiceMice, Inbred BALB CMicroscopy, Electron, TransmissionMycobacterium Infections, NontuberculousMycobacterium ulceransPhagocytosisSkin Diseases, BacterialSkin Ulcer1099489 bytesliteraturehttp://purl.org/coar/resource_type/c_6501journal articlehttp://purl.org/coar/access_right/c_abf2application/pdffulltexthttps://prod-dspace.uminho.pt/bitstreams/a2a239e7-64ff-4186-90c3-df867fbc1361/download
spellingShingle Evidence for an intramacrophage growth phase of Mycobacterium ulcerans
Torrado, Egídio
Animals
Bacterial Toxins
Cells, Cultured
Disease Models, Animal
Female
Foot
Histocytochemistry
Humans
Macrolides
Macrophages
Mice
Mice, Inbred BALB C
Microscopy, Electron, Transmission
Mycobacterium Infections, Nontuberculous
Mycobacterium ulcerans
Phagocytosis
Skin Diseases, Bacterial
Skin Ulcer
status SINGLETON
subject.fl_str_mv Animals
Bacterial Toxins
Cells, Cultured
Disease Models, Animal
Female
Foot
Histocytochemistry
Humans
Macrolides
Macrophages
Mice
Mice, Inbred BALB C
Microscopy, Electron, Transmission
Mycobacterium Infections, Nontuberculous
Mycobacterium ulcerans
Phagocytosis
Skin Diseases, Bacterial
Skin Ulcer
title Evidence for an intramacrophage growth phase of Mycobacterium ulcerans
title_full Evidence for an intramacrophage growth phase of Mycobacterium ulcerans
title_fullStr Evidence for an intramacrophage growth phase of Mycobacterium ulcerans
title_full_unstemmed Evidence for an intramacrophage growth phase of Mycobacterium ulcerans
title_short Evidence for an intramacrophage growth phase of Mycobacterium ulcerans
title_sort Evidence for an intramacrophage growth phase of Mycobacterium ulcerans
topic Animals
Bacterial Toxins
Cells, Cultured
Disease Models, Animal
Female
Foot
Histocytochemistry
Humans
Macrolides
Macrophages
Mice
Mice, Inbred BALB C
Microscopy, Electron, Transmission
Mycobacterium Infections, Nontuberculous
Mycobacterium ulcerans
Phagocytosis
Skin Diseases, Bacterial
Skin Ulcer
topic_facet Animals
Bacterial Toxins
Cells, Cultured
Disease Models, Animal
Female
Foot
Histocytochemistry
Humans
Macrolides
Macrophages
Mice
Mice, Inbred BALB C
Microscopy, Electron, Transmission
Mycobacterium Infections, Nontuberculous
Mycobacterium ulcerans
Phagocytosis
Skin Diseases, Bacterial
Skin Ulcer
url https://hdl.handle.net/1822/67769
visible 1