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BRD9 status is a major contributor for cysteine metabolic remodeling through MST and EAAT3 modulation in malignant melanoma

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Resumo:Cutaneous melanoma (CM) is the most aggressive skin cancer, showing globally increasing incidence. Hereditary CM accounts for a significant percentage (5-15 %) of all CM cases. However, most familial cases remain without a known genetic cause. Even though, BRD9 has been associated to CM as a susceptibility gene. The molecular events following BRD9 mutagenesis are still not completely understood. In this study, we disclosed BRD9 as a key regulator in cysteine metabolism and associated altered BRD9 to increased cell proliferation, migration and invasiveness, as well as to altered melanin levels, inducing higher susceptibility to melanomagenesis. It is evident that BRD9 WT and mutated BRD9 (c.183G>C) have a different impact on cysteine metabolism, respectively by inhibiting and activating MPST expression in the metastatic A375 cell line. The effect of the mutated BRD9 variant was more evident in A375 cells than in the less invasive WM115 line. Our data point out novel molecular and metabolic mechanisms dependent on BRD9 status that potentially account for the increased risk of developing CM and enhancing CM aggressiveness. Moreover, our findings emphasize the role of cysteine metabolism remodeling in melanoma progression and open new queues to follow to explore the role of BRD9 as a melanoma susceptibility or cancer-related gene.
Autores principais:Hipólito, Ana
Outros Autores:Xavier, Renato; Brito, Cheila; Tomás, Ana; Lemos, Isabel; Cabaço, Luís C.; Cabaço, Luís C.; Silva, Fernanda; Oliva, Abel; Barral, Duarte C.; C. Barral, Duarte; Vicente, João B.; Gonçalves, Luís G.; Pojo, Marta; Serpa, Jacinta; Serpa, Jacinta
Assunto:BRD9 Cutaneous melanoma Cysteine metabolism Metabolic remodeling Molecular Medicine Molecular Biology SDG 3 - Good Health and Well-being
Ano:2024
País:Portugal
Tipo de documento:artigo
Tipo de acesso:acesso aberto
Instituição associada:Universidade Nova de Lisboa
Idioma:inglês
Origem:Repositório Institucional da UNL
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author Hipólito, Ana
author2 Xavier, Renato
Brito, Cheila
Tomás, Ana
Lemos, Isabel
Cabaço, Luís C.
Cabaço, Luís C.
Silva, Fernanda
Oliva, Abel
Barral, Duarte C.
C. Barral, Duarte
Vicente, João B.
Gonçalves, Luís G.
Pojo, Marta
Serpa, Jacinta
Serpa, Jacinta
author2_role author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author_facet Hipólito, Ana
Xavier, Renato
Brito, Cheila
Tomás, Ana
Lemos, Isabel
Cabaço, Luís C.
Cabaço, Luís C.
Silva, Fernanda
Oliva, Abel
Barral, Duarte C.
C. Barral, Duarte
Vicente, João B.
Gonçalves, Luís G.
Pojo, Marta
Serpa, Jacinta
Serpa, Jacinta
author_role author
contributor_name_str_mv NOVA Medical School|Faculdade de Ciências Médicas (NMS|FCM)
iNOVA4Health - pólo NMS
Instituto de Tecnologia Química e Biológica António Xavier (ITQB)
Elsevier
RUN
country_str PT
creators_json_txt [{\"Person.name\":\"Hipólito, Ana\"},{\"Person.name\":\"Xavier, Renato\"},{\"Person.name\":\"Brito, Cheila\"},{\"Person.name\":\"Tomás, Ana\"},{\"Person.name\":\"Lemos, Isabel\"},{\"Person.name\":\"Cabaço, Luís C.\"},{\"Person.name\":\"Cabaço, Luís C.\",\"Person.identifier.orcid\":\"0000-0003-0976-1781\"},{\"Person.name\":\"Silva, Fernanda\"},{\"Person.name\":\"Oliva, Abel\"},{\"Person.name\":\"Barral, Duarte C.\"},{\"Person.name\":\"C. Barral, Duarte\",\"Person.identifier.orcid\":\"0000-0001-8867-2407\"},{\"Person.name\":\"Vicente, João B.\"},{\"Person.name\":\"Gonçalves, Luís G.\"},{\"Person.name\":\"Pojo, Marta\"},{\"Person.name\":\"Serpa, Jacinta\"},{\"Person.name\":\"Serpa, Jacinta\",\"Person.identifier.orcid\":\"0000-0002-1548-5907\"}]
datacite.contributors.contributor.contributorName.fl_str_mv NOVA Medical School|Faculdade de Ciências Médicas (NMS|FCM)
iNOVA4Health - pólo NMS
Instituto de Tecnologia Química e Biológica António Xavier (ITQB)
Elsevier
RUN
datacite.creators.creator.creatorName.fl_str_mv Hipólito, Ana
Xavier, Renato
Brito, Cheila
Tomás, Ana
Lemos, Isabel
Cabaço, Luís C.
Cabaço, Luís C.
Silva, Fernanda
Oliva, Abel
Barral, Duarte C.
C. Barral, Duarte
Vicente, João B.
Gonçalves, Luís G.
Pojo, Marta
Serpa, Jacinta
Serpa, Jacinta
datacite.date.Accepted.fl_str_mv 2024-02-01T00:00:00Z
datacite.date.available.fl_str_mv 2024-01-17T22:27:14Z
datacite.date.embargoed.fl_str_mv 2024-01-17T22:27:14Z
datacite.rights.fl_str_mv http://purl.org/coar/access_right/c_abf2
datacite.subjects.subject.fl_str_mv BRD9
Cutaneous melanoma
Cysteine metabolism
Metabolic remodeling
Molecular Medicine
Molecular Biology
SDG 3 - Good Health and Well-being
datacite.titles.title.fl_str_mv BRD9 status is a major contributor for cysteine metabolic remodeling through MST and EAAT3 modulation in malignant melanoma
dc.contributor.none.fl_str_mv NOVA Medical School|Faculdade de Ciências Médicas (NMS|FCM)
iNOVA4Health - pólo NMS
Instituto de Tecnologia Química e Biológica António Xavier (ITQB)
Elsevier
RUN
dc.creator.none.fl_str_mv Hipólito, Ana
Xavier, Renato
Brito, Cheila
Tomás, Ana
Lemos, Isabel
Cabaço, Luís C.
Cabaço, Luís C.
Silva, Fernanda
Oliva, Abel
Barral, Duarte C.
C. Barral, Duarte
Vicente, João B.
Gonçalves, Luís G.
Pojo, Marta
Serpa, Jacinta
Serpa, Jacinta
dc.date.Accepted.fl_str_mv 2024-02-01T00:00:00Z
dc.date.available.fl_str_mv 2024-01-17T22:27:14Z
dc.date.embargoed.fl_str_mv 2024-01-17T22:27:14Z
dc.format.none.fl_str_mv application/pdf
dc.identifier.none.fl_str_mv http://hdl.handle.net/10362/162409
dc.language.none.fl_str_mv eng
dc.rights.none.fl_str_mv http://purl.org/coar/access_right/c_abf2
dc.subject.none.fl_str_mv BRD9
Cutaneous melanoma
Cysteine metabolism
Metabolic remodeling
Molecular Medicine
Molecular Biology
SDG 3 - Good Health and Well-being
dc.title.fl_str_mv BRD9 status is a major contributor for cysteine metabolic remodeling through MST and EAAT3 modulation in malignant melanoma
dc.type.none.fl_str_mv http://purl.org/coar/resource_type/c_6501
description Cutaneous melanoma (CM) is the most aggressive skin cancer, showing globally increasing incidence. Hereditary CM accounts for a significant percentage (5-15 %) of all CM cases. However, most familial cases remain without a known genetic cause. Even though, BRD9 has been associated to CM as a susceptibility gene. The molecular events following BRD9 mutagenesis are still not completely understood. In this study, we disclosed BRD9 as a key regulator in cysteine metabolism and associated altered BRD9 to increased cell proliferation, migration and invasiveness, as well as to altered melanin levels, inducing higher susceptibility to melanomagenesis. It is evident that BRD9 WT and mutated BRD9 (c.183G>C) have a different impact on cysteine metabolism, respectively by inhibiting and activating MPST expression in the metastatic A375 cell line. The effect of the mutated BRD9 variant was more evident in A375 cells than in the less invasive WM115 line. Our data point out novel molecular and metabolic mechanisms dependent on BRD9 status that potentially account for the increased risk of developing CM and enhancing CM aggressiveness. Moreover, our findings emphasize the role of cysteine metabolism remodeling in melanoma progression and open new queues to follow to explore the role of BRD9 as a melanoma susceptibility or cancer-related gene.
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eu_rights_str_mv openAccess
format article
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identifier.url.fl_str_mv http://hdl.handle.net/10362/162409
inst_facet_str urn:organizationAcronym:unl{{{_:::_}}}Universidade Nova de Lisboa
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institution Universidade Nova de Lisboa
instname_str Universidade Nova de Lisboa
language eng
network_acronym_str run
network_name_str Repositório Institucional da UNL
oai_identifier_str oai:run.unl.pt:10362/162409
organization_str_mv urn:organizationAcronym:unl
person_str_mv Hipólito, Ana
Xavier, Renato
Brito, Cheila
Tomás, Ana
Lemos, Isabel
Cabaço, Luís C.
Cabaço, Luís C.
Cabaço, Luís C.
https://www.ciencia-id.pt/6D1D-EC36-6BA3
6D1D-EC36-6BA3
http://orcid.org/0000-0003-0976-1781
0000-0003-0976-1781
Silva, Fernanda
Oliva, Abel
Barral, Duarte C.
C. Barral, Duarte
C. Barral, Duarte
https://www.ciencia-id.pt/8810-DEBC-025E
8810-DEBC-025E
http://orcid.org/0000-0001-8867-2407
0000-0001-8867-2407
Vicente, João B.
Gonçalves, Luís G.
Pojo, Marta
Serpa, Jacinta
Serpa, Jacinta
Serpa, Jacinta
https://www.ciencia-id.pt/2610-7C0D-38F2
2610-7C0D-38F2
http://orcid.org/0000-0002-1548-5907
0000-0002-1548-5907
publishDate 2024
repo_facet_str urn:repositoryAcronym:run{{{_:::_}}}Repositório Institucional da UNL
reponame_str Repositório Institucional da UNL
repository_id_str urn:repositoryAcronym:run
service_str_mv urn:repositoryAcronym:run
spelling engenCutaneous melanoma (CM) is the most aggressive skin cancer, showing globally increasing incidence. Hereditary CM accounts for a significant percentage (5-15 %) of all CM cases. However, most familial cases remain without a known genetic cause. Even though, BRD9 has been associated to CM as a susceptibility gene. The molecular events following BRD9 mutagenesis are still not completely understood. In this study, we disclosed BRD9 as a key regulator in cysteine metabolism and associated altered BRD9 to increased cell proliferation, migration and invasiveness, as well as to altered melanin levels, inducing higher susceptibility to melanomagenesis. It is evident that BRD9 WT and mutated BRD9 (c.183G>C) have a different impact on cysteine metabolism, respectively by inhibiting and activating MPST expression in the metastatic A375 cell line. The effect of the mutated BRD9 variant was more evident in A375 cells than in the less invasive WM115 line. Our data point out novel molecular and metabolic mechanisms dependent on BRD9 status that potentially account for the increased risk of developing CM and enhancing CM aggressiveness. Moreover, our findings emphasize the role of cysteine metabolism remodeling in melanoma progression and open new queues to follow to explore the role of BRD9 as a melanoma susceptibility or cancer-related gene.application/pdfenBRD9 status is a major contributor for cysteine metabolic remodeling through MST and EAAT3 modulation in malignant melanomaHipólito, AnaXavier, RenatoBrito, CheilaTomás, AnaLemos, IsabelCabaço, Luís C.PersonalCabaço, Luís C.DSpacehttp://dspace.org/items/b545a125-c8cb-43af-a0c2-8b4dd1f7ee5fDSpacehttp://dspace.org/items/b545a125-c8cb-43af-a0c2-8b4dd1f7ee5fCabaçoLuís C.Ciência IDhttps://www.ciencia-id.pt6D1D-EC36-6BA3ORCIDhttp://orcid.org0000-0003-0976-1781Scopus Author IDhttps://www.scopus.com57224771399Silva, FernandaOliva, AbelBarral, Duarte C.PersonalC. Barral, DuarteDSpacehttp://dspace.org/items/e8205a0a-7fb2-450a-9917-417ec75d88b3DSpacehttp://dspace.org/items/e8205a0a-7fb2-450a-9917-417ec75d88b3BarralDuarteCiência IDhttps://www.ciencia-id.pt8810-DEBC-025EORCIDhttp://orcid.org0000-0001-8867-2407Researcher IDhttps://www.researcherid.comE-7548-2013Scopus Author IDhttps://www.scopus.com6602513159Scopus Author IDhttps://www.scopus.com6602513159Vicente, João B.Gonçalves, Luís G.Pojo, MartaSerpa, JacintaPersonalSerpa, JacintaDSpacehttp://dspace.org/items/5f6eaca2-f01f-4894-8da2-688671913214DSpacehttp://dspace.org/items/5f6eaca2-f01f-4894-8da2-688671913214SerpaJacintaCiência IDhttps://www.ciencia-id.pt2610-7C0D-38F2ORCIDhttp://orcid.org0000-0002-1548-5907Researcher IDhttps://www.researcherid.comO-8942-2014Scopus Author IDhttps://www.scopus.com6601922586Scopus Author IDhttps://www.scopus.com56623388500Scopus Author IDhttps://www.scopus.com56623388500NOVA Medical School|Faculdade de Ciências Médicas (NMS|FCM)iNOVA4Health - pólo NMSInstituto de Tecnologia Química e Biológica António Xavier (ITQB)ElsevierHostingInstitutionOrganizationalRUNe-mailmailto:run@unl.ptrun@unl.ptISSNIsPartOf1879-260XURNIsPartOfPURE: 81028392URNIsPartOfPURE UUID: 840b30bb-52c6-42da-96cc-1c144261dd44URNIsPartOfScopus: 85181396377URNIsPartOfPubMed: 38070581URNIsPartOfORCID: 0000-0001-8867-2407/work/149584570URNIsPartOfORCID: /0000-0001-8867-2407/work/151365894URNIsPartOfORCID: /0000-0001-5683-3552/work/151405763URNIsPartOfORCID: /0000-0002-1548-5907/work/151423015URNIsPartOfWOS: 001137400700001DOIIsPartOf10.1016/j.bbadis.2023.1669832024-01-17T22:27:14Z2024-02-012024-02-01T00:00:00ZHandlehttp://hdl.handle.net/10362/162409http://purl.org/coar/access_right/c_abf2open accessBRD9Cutaneous melanomaCysteine metabolismMetabolic remodelingMolecular MedicineMolecular BiologySDG 3 - Good Health and Well-being11992259 bytesliteraturehttp://purl.org/coar/resource_type/c_6501journal articlehttp://purl.org/coar/access_right/c_abf2application/pdffulltexthttps://run.unl.pt/bitstreams/e50c3de5-618a-498c-bb63-20c76d568c68/download
spellingShingle BRD9 status is a major contributor for cysteine metabolic remodeling through MST and EAAT3 modulation in malignant melanoma
Hipólito, Ana
BRD9
Cutaneous melanoma
Cysteine metabolism
Metabolic remodeling
Molecular Medicine
Molecular Biology
SDG 3 - Good Health and Well-being
status SINGLETON
subject.fl_str_mv BRD9
Cutaneous melanoma
Cysteine metabolism
Metabolic remodeling
Molecular Medicine
Molecular Biology
SDG 3 - Good Health and Well-being
title BRD9 status is a major contributor for cysteine metabolic remodeling through MST and EAAT3 modulation in malignant melanoma
title_full BRD9 status is a major contributor for cysteine metabolic remodeling through MST and EAAT3 modulation in malignant melanoma
title_fullStr BRD9 status is a major contributor for cysteine metabolic remodeling through MST and EAAT3 modulation in malignant melanoma
title_full_unstemmed BRD9 status is a major contributor for cysteine metabolic remodeling through MST and EAAT3 modulation in malignant melanoma
title_short BRD9 status is a major contributor for cysteine metabolic remodeling through MST and EAAT3 modulation in malignant melanoma
title_sort BRD9 status is a major contributor for cysteine metabolic remodeling through MST and EAAT3 modulation in malignant melanoma
topic BRD9
Cutaneous melanoma
Cysteine metabolism
Metabolic remodeling
Molecular Medicine
Molecular Biology
SDG 3 - Good Health and Well-being
topic_facet BRD9
Cutaneous melanoma
Cysteine metabolism
Metabolic remodeling
Molecular Medicine
Molecular Biology
SDG 3 - Good Health and Well-being
url http://hdl.handle.net/10362/162409
visible 1