Publicação

Can cryoglobulinemia trigger ANCA vasculitis?

Ver documento

Detalhes bibliográficos
Resumo:Antineutrophil cytoplasmic antibody (ANCA)-associated vasculitis is a rare and heterogeneous group of autoimmune diseases. These pauciimmune vasculitis differ from the cryoglobulinemic vasculitis mediated by immune complexes. We report the case of a 79-year-old male with silicosis and chronic alcoholic liver disease, presenting with a rapidly progressive glomerulonephritis and pancytopenia. Work-up revealed hypocomplementemia, polyclonal hypergammaglobulinemia, type 3 cryoglobulinemia, p-ANCA positivity and elevated anti-MPO. Renal biopsy showed a pattern of chronic interstitial nephritis with fibrocellular crescents, and immunofluorescence staining was negative. Treatment was started with corticosteroids and rituximab with improvement of the renal function, decrease of the anti-MPO titer and disappearance of the cryoglobulinemia. In this case, renal injury was caused by ANCA vasculitis, whose etiology remains unknown despite the recognizable risk factor for ANCA formation (silicosis). The importance of the cryoglobulinemia is not clear, as it could be part of the pathogenesis or just an epiphenomenon secondary to the autoimmune and the chronic liver diseases.
Autores principais:Cunha,Ivo
Outros Autores:Santos,M. Teresa; Pereira,P. Ricardo
Assunto:Crescents cryoglobulinemia interstitial nephritis rapidly progressive glomerulonephritis vasculitis
Ano:2020
País:Portugal
Tipo de documento:relatório
Tipo de acesso:acesso aberto
Instituição associada:Fundação para a Ciência e Tecnologia
Idioma:inglês
Origem:SciELO Portugal
_version_ 1868441695742328832
author Cunha,Ivo
author2 Santos,M. Teresa
Pereira,P. Ricardo
author2_role author
author
author_facet Cunha,Ivo
Santos,M. Teresa
Pereira,P. Ricardo
author_role author
country_str PT
creators_json_txt [{\"Person.name\":\"Cunha,Ivo\"},{\"Person.name\":\"Santos,M. Teresa\"},{\"Person.name\":\"Pereira,P. Ricardo\"}]
datacite.creators.creator.creatorName.fl_str_mv Cunha,Ivo
Santos,M. Teresa
Pereira,P. Ricardo
datacite.rights.fl_str_mv http://purl.org/coar/access_right/c_abf2
datacite.subjects.subject.fl_str_mv Crescents
cryoglobulinemia
interstitial nephritis
rapidly progressive glomerulonephritis
vasculitis
datacite.titles.title.fl_str_mv Can cryoglobulinemia trigger ANCA vasculitis?
dc.creator.none.fl_str_mv Cunha,Ivo
Santos,M. Teresa
Pereira,P. Ricardo
dc.format.none.fl_str_mv text/html
dc.identifier.none.fl_str_mv http://scielo.pt/scielo.php?script=sci_arttext&pid=S0872-01692020000100009
dc.language.none.fl_str_mv eng
dc.publisher.none.fl_str_mv Sociedade Portuguesa de Nefrologia
dc.rights.none.fl_str_mv http://purl.org/coar/access_right/c_abf2
dc.source.none.fl_str_mv Portuguese Journal of Nephrology & Hypertension v.34 n.1 2020
dc.subject.none.fl_str_mv Crescents
cryoglobulinemia
interstitial nephritis
rapidly progressive glomerulonephritis
vasculitis
dc.title.fl_str_mv Can cryoglobulinemia trigger ANCA vasculitis?
dc.type.none.fl_str_mv http://purl.org/coar/resource_type/c_93fc
description Antineutrophil cytoplasmic antibody (ANCA)-associated vasculitis is a rare and heterogeneous group of autoimmune diseases. These pauciimmune vasculitis differ from the cryoglobulinemic vasculitis mediated by immune complexes. We report the case of a 79-year-old male with silicosis and chronic alcoholic liver disease, presenting with a rapidly progressive glomerulonephritis and pancytopenia. Work-up revealed hypocomplementemia, polyclonal hypergammaglobulinemia, type 3 cryoglobulinemia, p-ANCA positivity and elevated anti-MPO. Renal biopsy showed a pattern of chronic interstitial nephritis with fibrocellular crescents, and immunofluorescence staining was negative. Treatment was started with corticosteroids and rituximab with improvement of the renal function, decrease of the anti-MPO titer and disappearance of the cryoglobulinemia. In this case, renal injury was caused by ANCA vasculitis, whose etiology remains unknown despite the recognizable risk factor for ANCA formation (silicosis). The importance of the cryoglobulinemia is not clear, as it could be part of the pathogenesis or just an epiphenomenon secondary to the autoimmune and the chronic liver diseases.
dirty 0
eu_rights_str_mv openAccess
format report
id scielopt_daf4474033efc4ff6567b4f2e97cc3da
identifier.url.fl_str_mv http://scielo.pt/scielo.php?script=sci_arttext&pid=S0872-01692020000100009
instacron_str SciELO
institution Fundação para a Ciência e Tecnologia
instname_str Fundação para a Ciência e Tecnologia
language eng
network_acronym_str scielopt
network_name_str SciELO Portugal
oai_identifier_str oai:scielo:S0872-01692020000100009
organization_str_mv urn:organizationAcronym:scielo
person_str_mv Cunha,Ivo
Santos,M. Teresa
Pereira,P. Ricardo
publishDate 2020
publisher.none.fl_str_mv Sociedade Portuguesa de Nefrologia
reponame_str SciELO Portugal
repository_id_str urn:repositoryAcronym:scielopt
service_str_mv urn:repositoryAcronym:scielopt
spelling Can cryoglobulinemia trigger ANCA vasculitis?Cunha,IvoSantos,M. TeresaPereira,P. RicardoCrescentscryoglobulinemiainterstitial nephritisrapidly progressive glomerulonephritisvasculitisopen accesshttp://purl.org/coar/access_right/c_abf2http://scielo.pt/scielo.php?script=sci_arttext&pid=S0872-01692020000100009URLhttp://scielo.pt/scielo.php?script=sci_arttext&pid=S0872-01692020000100009URLHasVersion2020-03-01Antineutrophil cytoplasmic antibody (ANCA)-associated vasculitis is a rare and heterogeneous group of autoimmune diseases. These pauciimmune vasculitis differ from the cryoglobulinemic vasculitis mediated by immune complexes. We report the case of a 79-year-old male with silicosis and chronic alcoholic liver disease, presenting with a rapidly progressive glomerulonephritis and pancytopenia. Work-up revealed hypocomplementemia, polyclonal hypergammaglobulinemia, type 3 cryoglobulinemia, p-ANCA positivity and elevated anti-MPO. Renal biopsy showed a pattern of chronic interstitial nephritis with fibrocellular crescents, and immunofluorescence staining was negative. Treatment was started with corticosteroids and rituximab with improvement of the renal function, decrease of the anti-MPO titer and disappearance of the cryoglobulinemia. In this case, renal injury was caused by ANCA vasculitis, whose etiology remains unknown despite the recognizable risk factor for ANCA formation (silicosis). The importance of the cryoglobulinemia is not clear, as it could be part of the pathogenesis or just an epiphenomenon secondary to the autoimmune and the chronic liver diseases.Sociedade Portuguesa de NefrologiaPortuguese Journal of Nephrology & Hypertension v.34 n.1 2020text/htmlengreporthttp://purl.org/coar/resource_type/c_93fcother research product
spellingShingle Can cryoglobulinemia trigger ANCA vasculitis?
Cunha,Ivo
Crescents
cryoglobulinemia
interstitial nephritis
rapidly progressive glomerulonephritis
vasculitis
status SINGLETON
subject.fl_str_mv Crescents
cryoglobulinemia
interstitial nephritis
rapidly progressive glomerulonephritis
vasculitis
title Can cryoglobulinemia trigger ANCA vasculitis?
title_full Can cryoglobulinemia trigger ANCA vasculitis?
title_fullStr Can cryoglobulinemia trigger ANCA vasculitis?
title_full_unstemmed Can cryoglobulinemia trigger ANCA vasculitis?
title_short Can cryoglobulinemia trigger ANCA vasculitis?
title_sort Can cryoglobulinemia trigger ANCA vasculitis?
topic Crescents
cryoglobulinemia
interstitial nephritis
rapidly progressive glomerulonephritis
vasculitis
topic_facet Crescents
cryoglobulinemia
interstitial nephritis
rapidly progressive glomerulonephritis
vasculitis
url http://scielo.pt/scielo.php?script=sci_arttext&pid=S0872-01692020000100009
visible 1