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Effects of oxygen depletion on transmembrane protein activities

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Resumo:Effects of oxygen depletion on cellular membranes are still poorly understood. Amphiphilic molecules are known to modulate the plasma membrane lipid bilayer’s physical properties; in turn, mechanical properties of the lipid bilayer affect signal transduction through numerous mechanosensitive transmembrane proteins including ion channels, receptor tyrosine kinases, NADPH oxidases and G-protein coupled receptors. Thus, the concentration of oxygen in/at the lipid bilayer may modulate its mechanical properties. Here we propose that: (i) under hypoxia, the plasma membrane lipid bilayer would become oxygen depleted, (ii) depletion of oxygen molecules might induce mechanical stress in the lipid bilayer, and (iii) hypoxia-induced mechanical stress in the lipid bilayer activates mechanosensitive transmembrane proteins and downstream signaling pathways. We provide evidence – on the basis of published experimental data – that there can be links between oxygen depletioninduced mechanical stress in the membrane and activation of some mechanisms participating in oxygen sensing, including reactive oxygen species (ROS) produced by mitochondrial complex III, ROS generated at the plasma membrane by NADPH oxidases, ion channels of the transient receptor potential family and increase in intracellular Ca2+ and stabilization of hypoxia-inducible factor 1α (HIF-1α).
Autores principais:Barvitenko, Nadezhda N.
Outros Autores:Aslam, Muhammad; Lawen, Alfons; Pantaleo, Antonella; Saldanha, Carlota; Matteucci, Elena
Assunto:Oxygen, hypoxia Mechanical stress Hypoxia inducible factor Mitochondria Ion channel G-protein coupled receptor
Ano:2015
País:Portugal
Tipo de documento:artigo
Tipo de acesso:acesso restrito
Instituição associada:Universidade de Lisboa
Idioma:inglês
Origem:Repositório da Universidade de Lisboa
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author Barvitenko, Nadezhda N.
author2 Aslam, Muhammad
Lawen, Alfons
Pantaleo, Antonella
Saldanha, Carlota
Matteucci, Elena
author2_role author
author
author
author
author
author_facet Barvitenko, Nadezhda N.
Aslam, Muhammad
Lawen, Alfons
Pantaleo, Antonella
Saldanha, Carlota
Matteucci, Elena
author_role author
contributor_name_str_mv Repositório Científico de Acesso Aberto da ULisboa
country_str PT
creators_json_txt [{\"Person.name\":\"Barvitenko, Nadezhda N.\"},{\"Person.name\":\"Aslam, Muhammad\"},{\"Person.name\":\"Lawen, Alfons\"},{\"Person.name\":\"Pantaleo, Antonella\"},{\"Person.name\":\"Saldanha, Carlota\"},{\"Person.name\":\"Matteucci, Elena\"}]
datacite.contributors.contributor.contributorName.fl_str_mv Repositório Científico de Acesso Aberto da ULisboa
datacite.creators.creator.creatorName.fl_str_mv Barvitenko, Nadezhda N.
Aslam, Muhammad
Lawen, Alfons
Pantaleo, Antonella
Saldanha, Carlota
Matteucci, Elena
datacite.date.Accepted.fl_str_mv 2015-01-01T00:00:00Z
datacite.date.available.fl_str_mv 2018-07-20T10:37:44Z
datacite.date.embargoed.fl_str_mv 2018-07-20T10:37:44Z
datacite.rights.fl_str_mv http://purl.org/coar/access_right/c_16ec
datacite.subjects.subject.fl_str_mv Oxygen, hypoxia
Mechanical stress
Hypoxia inducible factor
Mitochondria
Ion channel
G-protein coupled receptor
datacite.titles.title.fl_str_mv Effects of oxygen depletion on transmembrane protein activities
dc.contributor.none.fl_str_mv Repositório Científico de Acesso Aberto da ULisboa
dc.creator.none.fl_str_mv Barvitenko, Nadezhda N.
Aslam, Muhammad
Lawen, Alfons
Pantaleo, Antonella
Saldanha, Carlota
Matteucci, Elena
dc.date.Accepted.fl_str_mv 2015-01-01T00:00:00Z
dc.date.available.fl_str_mv 2018-07-20T10:37:44Z
dc.date.embargoed.fl_str_mv 2018-07-20T10:37:44Z
dc.format.none.fl_str_mv application/pdf
dc.identifier.none.fl_str_mv http://hdl.handle.net/10451/34263
dc.language.none.fl_str_mv eng
dc.publisher.none.fl_str_mv Bentham Science Publishers
dc.rights.none.fl_str_mv http://purl.org/coar/access_right/c_16ec
dc.subject.none.fl_str_mv Oxygen, hypoxia
Mechanical stress
Hypoxia inducible factor
Mitochondria
Ion channel
G-protein coupled receptor
dc.title.fl_str_mv Effects of oxygen depletion on transmembrane protein activities
dc.type.none.fl_str_mv http://purl.org/coar/resource_type/c_6501
description Effects of oxygen depletion on cellular membranes are still poorly understood. Amphiphilic molecules are known to modulate the plasma membrane lipid bilayer’s physical properties; in turn, mechanical properties of the lipid bilayer affect signal transduction through numerous mechanosensitive transmembrane proteins including ion channels, receptor tyrosine kinases, NADPH oxidases and G-protein coupled receptors. Thus, the concentration of oxygen in/at the lipid bilayer may modulate its mechanical properties. Here we propose that: (i) under hypoxia, the plasma membrane lipid bilayer would become oxygen depleted, (ii) depletion of oxygen molecules might induce mechanical stress in the lipid bilayer, and (iii) hypoxia-induced mechanical stress in the lipid bilayer activates mechanosensitive transmembrane proteins and downstream signaling pathways. We provide evidence – on the basis of published experimental data – that there can be links between oxygen depletioninduced mechanical stress in the membrane and activation of some mechanisms participating in oxygen sensing, including reactive oxygen species (ROS) produced by mitochondrial complex III, ROS generated at the plasma membrane by NADPH oxidases, ion channels of the transient receptor potential family and increase in intracellular Ca2+ and stabilization of hypoxia-inducible factor 1α (HIF-1α).
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eu_rights_str_mv restrictedAccess
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fulltext.url.fl_str_mv https://repositorio.ulisboa.pt/bitstreams/5a545d32-c48e-4a32-a0b6-53109358ac3f/download
id ul_034f6c3cf8f91fcd38ce2c425bd2a84b
identifier.url.fl_str_mv http://hdl.handle.net/10451/34263
instacron_str ul
institution Universidade de Lisboa
instname_str Universidade de Lisboa
language eng
network_acronym_str ul
network_name_str Repositório da Universidade de Lisboa
oai_identifier_str oai:repositorio.ulisboa.pt:10451/34263
organization_str_mv urn:organizationAcronym:ul
person_str_mv Barvitenko, Nadezhda N.
Aslam, Muhammad
Lawen, Alfons
Pantaleo, Antonella
Saldanha, Carlota
Matteucci, Elena
publishDate 2015
publisher.none.fl_str_mv Bentham Science Publishers
reponame_str Repositório da Universidade de Lisboa
repository_id_str urn:repositoryAcronym:ul
service_str_mv urn:repositoryAcronym:ul
spelling engBentham Science Publisherspt_PTEffects of oxygen depletion on cellular membranes are still poorly understood. Amphiphilic molecules are known to modulate the plasma membrane lipid bilayer’s physical properties; in turn, mechanical properties of the lipid bilayer affect signal transduction through numerous mechanosensitive transmembrane proteins including ion channels, receptor tyrosine kinases, NADPH oxidases and G-protein coupled receptors. Thus, the concentration of oxygen in/at the lipid bilayer may modulate its mechanical properties. Here we propose that: (i) under hypoxia, the plasma membrane lipid bilayer would become oxygen depleted, (ii) depletion of oxygen molecules might induce mechanical stress in the lipid bilayer, and (iii) hypoxia-induced mechanical stress in the lipid bilayer activates mechanosensitive transmembrane proteins and downstream signaling pathways. We provide evidence – on the basis of published experimental data – that there can be links between oxygen depletioninduced mechanical stress in the membrane and activation of some mechanisms participating in oxygen sensing, including reactive oxygen species (ROS) produced by mitochondrial complex III, ROS generated at the plasma membrane by NADPH oxidases, ion channels of the transient receptor potential family and increase in intracellular Ca2+ and stabilization of hypoxia-inducible factor 1α (HIF-1α).application/pdfpt_PTEffects of oxygen depletion on transmembrane protein activitiesBarvitenko, Nadezhda N.Aslam, MuhammadLawen, AlfonsPantaleo, AntonellaSaldanha, CarlotaMatteucci, ElenaHostingInstitutionOrganizationalRepositório Científico de Acesso Aberto da ULisboae-mailmailto:repositorio@reitoria.ulisboa.ptrepositorio@reitoria.ulisboa.ptISSNIsPartOf1385-2728DOIIsPartOf10.2174/13852728196661507131800312018-07-20T10:37:44Z20152015-01-01T00:00:00ZHandlehttp://hdl.handle.net/10451/34263http://purl.org/coar/access_right/c_16ecrestricted accessOxygen, hypoxiaMechanical stressHypoxia inducible factorMitochondriaIon channelG-protein coupled receptor442636 bytesliteraturehttp://purl.org/coar/resource_type/c_6501journal articlehttp://purl.org/coar/access_right/c_16ecapplication/pdffulltexthttps://repositorio.ulisboa.pt/bitstreams/5a545d32-c48e-4a32-a0b6-53109358ac3f/downloadCurrent Organic Chemistry192020022010
spellingShingle Effects of oxygen depletion on transmembrane protein activities
Barvitenko, Nadezhda N.
Oxygen, hypoxia
Mechanical stress
Hypoxia inducible factor
Mitochondria
Ion channel
G-protein coupled receptor
status SINGLETON
subject.fl_str_mv Oxygen, hypoxia
Mechanical stress
Hypoxia inducible factor
Mitochondria
Ion channel
G-protein coupled receptor
title Effects of oxygen depletion on transmembrane protein activities
title_full Effects of oxygen depletion on transmembrane protein activities
title_fullStr Effects of oxygen depletion on transmembrane protein activities
title_full_unstemmed Effects of oxygen depletion on transmembrane protein activities
title_short Effects of oxygen depletion on transmembrane protein activities
title_sort Effects of oxygen depletion on transmembrane protein activities
topic Oxygen, hypoxia
Mechanical stress
Hypoxia inducible factor
Mitochondria
Ion channel
G-protein coupled receptor
topic_facet Oxygen, hypoxia
Mechanical stress
Hypoxia inducible factor
Mitochondria
Ion channel
G-protein coupled receptor
url http://hdl.handle.net/10451/34263
visible 1