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Primary ciliary dyskinesia due to CCNO mutations : a genotype‐phenotype correlation contribution

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Resumo:Primary ciliary dyskinesia (PCD) is genetically and clinically heterogeneous. CCNO mutations are associated with chronic destructive lung disease and were first described in 2014. Early reports suggest that CCNO is mutated more frequently than expected, however, these are considered rare. We report on three eleven-year-old children with PCD due to CCNO mutations. All children presented early-onset respiratory symptoms, no cardiac or situs anomalies and moderate to severe clinical courses. Patients 1 and 3 were admitted to a neonatal intensive care unit due to respiratory distress. Patients 1 and 2 had atelectasis and lobar collapse, for which lobectomy was performed for patient 1. Patient 3 also presented otitis media with effusion with conductive hearing loss, requiring tympanostomy tube insertion twice. Diagnosis of PCD for all three required repeated nasal brushings, delaying diagnostic confirmation. Microscopy analysis revealed severely decreased numbers of cilia, but normal ultrastructure and uncoordinated beat pattern in the residual cilia. Surprisingly, the prevalence of pathogenic CCNO variants in our centre is higher than expected (three out of sixteen patients). Pathogenic variants in PCD-causing genes lead to specific ultrastructural defects, and there is a suggestion for genotype-phenotype association. However, there are little longitudinal data evaluating the impact of specific defects on disease progression, but a recent study showed a worse lung disease and poorer nutritional status. Concluding, this report underlies the importance of patient-oriented diagnosis and management in highly experienced PCD centres.
Autores principais:Henriques, Ana Raquel
Outros Autores:Constant, Carolina; Descalço, Andreia; Pinto, Andreia; Moura Nunes, J.; Sampaio, Pedro; Lopes, Susana S.; Pereira, Luísa; Bandeira, Teresa
Assunto:CCNO mutations Child Chronic diseases Ciliary motility disorders
Ano:2021
País:Portugal
Tipo de documento:artigo
Tipo de acesso:acesso restrito
Instituição associada:Universidade de Lisboa
Idioma:inglês
Origem:Repositório da Universidade de Lisboa
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author Henriques, Ana Raquel
author2 Constant, Carolina
Descalço, Andreia
Pinto, Andreia
Moura Nunes, J.
Sampaio, Pedro
Lopes, Susana S.
Pereira, Luísa
Bandeira, Teresa
author2_role author
author
author
author
author
author
author
author
author_facet Henriques, Ana Raquel
Constant, Carolina
Descalço, Andreia
Pinto, Andreia
Moura Nunes, J.
Sampaio, Pedro
Lopes, Susana S.
Pereira, Luísa
Bandeira, Teresa
author_role author
contributor_name_str_mv Repositório Científico de Acesso Aberto da ULisboa
country_str PT
creators_json_txt [{\"Person.name\":\"Henriques, Ana Raquel\"},{\"Person.name\":\"Constant, Carolina\",\"Person.identifier.orcid\":\"0000-0002-6596-1596\"},{\"Person.name\":\"Descalço, Andreia\"},{\"Person.name\":\"Pinto, Andreia\",\"Person.identifier.orcid\":\"0000-0002-0840-6844\"},{\"Person.name\":\"Moura Nunes, J.\"},{\"Person.name\":\"Sampaio, Pedro\"},{\"Person.name\":\"Lopes, Susana S.\"},{\"Person.name\":\"Pereira, Luísa\"},{\"Person.name\":\"Bandeira, Teresa\",\"Person.identifier.orcid\":\"0000-0002-6214-3117\"}]
datacite.contributors.contributor.contributorName.fl_str_mv Repositório Científico de Acesso Aberto da ULisboa
datacite.creators.creator.creatorName.fl_str_mv Henriques, Ana Raquel
Constant, Carolina
Descalço, Andreia
Pinto, Andreia
Moura Nunes, J.
Sampaio, Pedro
Lopes, Susana S.
Pereira, Luísa
Bandeira, Teresa
datacite.date.Accepted.fl_str_mv 2021-01-01T00:00:00Z
datacite.date.available.fl_str_mv 2021-06-21T16:47:12Z
datacite.date.embargoed.fl_str_mv 2021-06-21T16:47:12Z
datacite.rights.fl_str_mv http://purl.org/coar/access_right/c_16ec
datacite.subjects.subject.fl_str_mv CCNO mutations
Child
Chronic diseases
Ciliary motility disorders
datacite.titles.title.fl_str_mv Primary ciliary dyskinesia due to CCNO mutations : a genotype‐phenotype correlation contribution
dc.contributor.none.fl_str_mv Repositório Científico de Acesso Aberto da ULisboa
dc.creator.none.fl_str_mv Henriques, Ana Raquel
Constant, Carolina
Descalço, Andreia
Pinto, Andreia
Moura Nunes, J.
Sampaio, Pedro
Lopes, Susana S.
Pereira, Luísa
Bandeira, Teresa
dc.date.Accepted.fl_str_mv 2021-01-01T00:00:00Z
dc.date.available.fl_str_mv 2021-06-21T16:47:12Z
dc.date.embargoed.fl_str_mv 2021-06-21T16:47:12Z
dc.format.none.fl_str_mv application/pdf
dc.identifier.none.fl_str_mv http://hdl.handle.net/10451/48683
dc.language.none.fl_str_mv eng
dc.publisher.none.fl_str_mv John Wiley & Sons, Inc.
dc.rights.none.fl_str_mv http://purl.org/coar/access_right/c_16ec
dc.subject.none.fl_str_mv CCNO mutations
Child
Chronic diseases
Ciliary motility disorders
dc.title.fl_str_mv Primary ciliary dyskinesia due to CCNO mutations : a genotype‐phenotype correlation contribution
dc.type.none.fl_str_mv http://purl.org/coar/resource_type/c_6501
description Primary ciliary dyskinesia (PCD) is genetically and clinically heterogeneous. CCNO mutations are associated with chronic destructive lung disease and were first described in 2014. Early reports suggest that CCNO is mutated more frequently than expected, however, these are considered rare. We report on three eleven-year-old children with PCD due to CCNO mutations. All children presented early-onset respiratory symptoms, no cardiac or situs anomalies and moderate to severe clinical courses. Patients 1 and 3 were admitted to a neonatal intensive care unit due to respiratory distress. Patients 1 and 2 had atelectasis and lobar collapse, for which lobectomy was performed for patient 1. Patient 3 also presented otitis media with effusion with conductive hearing loss, requiring tympanostomy tube insertion twice. Diagnosis of PCD for all three required repeated nasal brushings, delaying diagnostic confirmation. Microscopy analysis revealed severely decreased numbers of cilia, but normal ultrastructure and uncoordinated beat pattern in the residual cilia. Surprisingly, the prevalence of pathogenic CCNO variants in our centre is higher than expected (three out of sixteen patients). Pathogenic variants in PCD-causing genes lead to specific ultrastructural defects, and there is a suggestion for genotype-phenotype association. However, there are little longitudinal data evaluating the impact of specific defects on disease progression, but a recent study showed a worse lung disease and poorer nutritional status. Concluding, this report underlies the importance of patient-oriented diagnosis and management in highly experienced PCD centres.
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id ul_613124fa4b8ded95dfe3ff8a48de4183
identifier.url.fl_str_mv http://hdl.handle.net/10451/48683
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institution Universidade de Lisboa
instname_str Universidade de Lisboa
language eng
network_acronym_str ul
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oai_identifier_str oai:repositorio.ulisboa.pt:10451/48683
organization_str_mv urn:organizationAcronym:ul
person_str_mv Henriques, Ana Raquel
Constant, Carolina
Constant, Carolina
http://orcid.org/0000-0002-6596-1596
0000-0002-6596-1596
Descalço, Andreia
Pinto, Andreia
Pinto, Andreia
http://orcid.org/0000-0002-0840-6844
0000-0002-0840-6844
Moura Nunes, J.
Sampaio, Pedro
Lopes, Susana S.
Pereira, Luísa
Bandeira, Teresa
Bandeira, Teresa
https://www.ciencia-id.pt/7B19-E545-226D
7B19-E545-226D
http://orcid.org/0000-0002-6214-3117
0000-0002-6214-3117
publishDate 2021
publisher.none.fl_str_mv John Wiley & Sons, Inc.
reponame_str Repositório da Universidade de Lisboa
repository_id_str urn:repositoryAcronym:ul
service_str_mv urn:repositoryAcronym:ul
spelling engJohn Wiley & Sons, Inc.pt_PTPrimary ciliary dyskinesia (PCD) is genetically and clinically heterogeneous. CCNO mutations are associated with chronic destructive lung disease and were first described in 2014. Early reports suggest that CCNO is mutated more frequently than expected, however, these are considered rare. We report on three eleven-year-old children with PCD due to CCNO mutations. All children presented early-onset respiratory symptoms, no cardiac or situs anomalies and moderate to severe clinical courses. Patients 1 and 3 were admitted to a neonatal intensive care unit due to respiratory distress. Patients 1 and 2 had atelectasis and lobar collapse, for which lobectomy was performed for patient 1. Patient 3 also presented otitis media with effusion with conductive hearing loss, requiring tympanostomy tube insertion twice. Diagnosis of PCD for all three required repeated nasal brushings, delaying diagnostic confirmation. Microscopy analysis revealed severely decreased numbers of cilia, but normal ultrastructure and uncoordinated beat pattern in the residual cilia. Surprisingly, the prevalence of pathogenic CCNO variants in our centre is higher than expected (three out of sixteen patients). Pathogenic variants in PCD-causing genes lead to specific ultrastructural defects, and there is a suggestion for genotype-phenotype association. However, there are little longitudinal data evaluating the impact of specific defects on disease progression, but a recent study showed a worse lung disease and poorer nutritional status. Concluding, this report underlies the importance of patient-oriented diagnosis and management in highly experienced PCD centres.application/pdfpt_PTPrimary ciliary dyskinesia due to CCNO mutations : a genotype‐phenotype correlation contributionHenriques, Ana RaquelPersonalConstant, CarolinaDSpacehttp://dspace.org/items/adcb84cf-9b1d-43ab-ab73-4d1be5881191DSpacehttp://dspace.org/items/adcb84cf-9b1d-43ab-ab73-4d1be5881191ConstantCarolinaORCIDhttp://orcid.org0000-0002-6596-1596Descalço, AndreiaPersonalPinto, AndreiaDSpacehttp://dspace.org/items/eae0f147-b206-4c61-9860-ff60215b9168DSpacehttp://dspace.org/items/eae0f147-b206-4c61-9860-ff60215b9168PintoAndreiaORCIDhttp://orcid.org0000-0002-0840-6844Moura Nunes, J.Sampaio, PedroLopes, Susana S.Pereira, LuísaPersonalBandeira, TeresaDSpacehttp://dspace.org/items/38518bb2-ba58-4ab9-8bab-c4f342f51123DSpacehttp://dspace.org/items/38518bb2-ba58-4ab9-8bab-c4f342f51123BandeiraTeresaCiência IDhttps://www.ciencia-id.pt7B19-E545-226DORCIDhttp://orcid.org0000-0002-6214-3117Researcher IDhttps://www.researcherid.comC-4397-2016Scopus Author IDhttps://www.scopus.com6507931000HostingInstitutionOrganizationalRepositório Científico de Acesso Aberto da ULisboae-mailmailto:repositorio@reitoria.ulisboa.ptrepositorio@reitoria.ulisboa.ptISSNIsPartOf8755-6863DOIIsPartOf10.1002/ppul.254402021-06-21T16:47:12Z20212021-01-01T00:00:00ZHandlehttp://hdl.handle.net/10451/48683http://purl.org/coar/access_right/c_16ecrestricted accessCCNO mutationsChildChronic diseasesCiliary motility disorders295414 bytesliteraturehttp://purl.org/coar/resource_type/c_6501journal articlehttp://purl.org/coar/access_right/c_16ecapplication/pdffulltexthttps://repositorio.ulisboa.pt/bitstreams/4b5c0993-b2d8-4e8f-aa4b-2ccb2131c09c/downloadPediatric Pulmonology
spellingShingle Primary ciliary dyskinesia due to CCNO mutations : a genotype‐phenotype correlation contribution
Henriques, Ana Raquel
CCNO mutations
Child
Chronic diseases
Ciliary motility disorders
status SINGLETON
subject.fl_str_mv CCNO mutations
Child
Chronic diseases
Ciliary motility disorders
title Primary ciliary dyskinesia due to CCNO mutations : a genotype‐phenotype correlation contribution
title_full Primary ciliary dyskinesia due to CCNO mutations : a genotype‐phenotype correlation contribution
title_fullStr Primary ciliary dyskinesia due to CCNO mutations : a genotype‐phenotype correlation contribution
title_full_unstemmed Primary ciliary dyskinesia due to CCNO mutations : a genotype‐phenotype correlation contribution
title_short Primary ciliary dyskinesia due to CCNO mutations : a genotype‐phenotype correlation contribution
title_sort Primary ciliary dyskinesia due to CCNO mutations : a genotype‐phenotype correlation contribution
topic CCNO mutations
Child
Chronic diseases
Ciliary motility disorders
topic_facet CCNO mutations
Child
Chronic diseases
Ciliary motility disorders
url http://hdl.handle.net/10451/48683
visible 1